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Sources

Experiment + Assay

Phenotypes

Human Diseases

Fig. 5E

Naylor RW et al. (2009)

Xla Wt + cdknx (Xic1)

NF20 (immunohistochemistry)

Anatomical Phenotype
abnormally decreased number of cell decreased size of the pronephric kidney obsolete abnormal histone phosphorylation

Fig. 5B

Naylor RW et al. (2009)

Xla Wt + cdknx (Xic1)

NF20 (in situ hybridization)

Expression Phenotype
decreased amount lhx1.L expression in pronephric mesenchyme

Fig. 4D

Naylor RW et al. (2009)

Xla Wt + cdknx (Xic1)

NF22 (in situ hybridization)

Anatomical Phenotype
whole organism abnormally curved dorsally

Expression Phenotype
absent lhx1.L expression in pronephric mesenchyme

FIG. 3C

Naylor RW et al. (2009)

Xla Wt + cdknx (Xic1)

NF32 (in situ hybridization)

Expression Phenotype
decreased amount nphs1.L expression in glomus

Fig. 2B

Naylor RW et al. (2009)

Xla Wt + cdknx (Xic1)

NF41 (immunohistochemistry)

Anatomical Phenotype
abnormal pronephric kidney abnormal pronephric tubule morphology decreased size of the distal tubule decreased size of the proximal tubule