| Search Criteria |
| Gene/Clone | Species | Stage | Anatomy Item | Experimenter |
|---|---|---|---|---|
| foxa1 | xenopus |
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Experiment details for foxa1
Ptbp1 and Exosc9 knockdowns trigger skin stability defects through different pathways.Ptbp1 and Exosc9 knockdowns trigger skin stability defects through different pathways.
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| Fig. 2. In situ hybridization for epidermal markers in exosc9 and ptbp1 morphants. Albino embryos were injected with the control morpholino (MoCo, A, D, G, J), MoPtbp1 (B, E, H, K) or MoExosc9 (C, F, I, L) in both blastomeres. Embryos were fixed at stage 32 and processed for in situ hybridization with the following probes: tuba1a (A–C), foxa1 (D–F), foxi1 (G–I) and itln2 (J–L). |