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Extended Cleavage Specificity of Human Neutrophil Elastase, Human Proteinase 3, and Their Distant Ortholog Clawed Frog PR3-Three Elastases With Similar Primary but Different Extended Specificities and Stability. , Fu Z, Thorpe M, Akula S, Chahal G, Hellman LT., Front Immunol. September 12, 2018; 9 2387.
Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 ( TRPV4) to Cause Inflammation and Pain. , Zhao P, Lieu T, Barlow N, Sostegni S, Haerteis S, Korbmacher C, Liedtke W, Jimenez-Vargas NN, Vanner SJ, Bunnett NW., J Biol Chem. May 29, 2015; 290 (22): 13875-87.
Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase. , Sostegni S, Diakov A, McIntyre P, Bunnett N, Korbmacher C, Haerteis S., Pflugers Arch. April 1, 2015; 467 (4): 687-701.
Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S. , Haerteis S, Krappitz M, Bertog M, Krappitz A, Baraznenok V, Henderson I, Lindström E, Murphy JE, Bunnett NW, Korbmacher C., Pflugers Arch. October 1, 2012; 464 (4): 353-65.
Plasmin and chymotrypsin have distinct preferences for channel activating cleavage sites in the γ subunit of the human epithelial sodium channel. , Haerteis S, Krappitz M, Diakov A, Krappitz A, Rauh R, Korbmacher C., J Gen Physiol. October 1, 2012; 140 (4): 375-89.
Cleavage in the {gamma}-subunit of the epithelial sodium channel (ENaC) plays an important role in the proteolytic activation of near-silent channels. , Diakov A, Bera K, Mokrushina M, Krueger B, Korbmacher C., J Physiol. October 1, 2008; 586 (19): 4587-608.
A novel neutrophil elastase inhibitor prevents elastase activation and surface cleavage of the epithelial sodium channel expressed in Xenopus laevis oocytes. , Harris M, Firsov D, Vuagniaux G, Stutts MJ, Rossier BC., J Biol Chem. January 5, 2007; 282 (1): 58-64.