Papers associated with kcnq3

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	Functional expression of two KvLQT1-related potassium channels responsible for an inherited idiopathic epilepsy., Yang WP, Levesque PC, Little WA, Conder ML, Ramakrishnan P, Neubauer MG, Blanar MA., J Biol Chem. July 31, 1998; 273 (31): 19419-23.
	KCNQ2 and KCNQ3 potassium channel subunits: molecular correlates of the M-channel., Wang HS, Pan Z, Shi W, Brown BS, Wymore RS, Cohen IS, Dixon JE, McKinnon D., Science. December 4, 1998; 282 (5395): 1890-3.
	Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy., Schroeder BC, Kubisch C, Stein V, Jentsch TJ., Nature. December 17, 1998; 396 (6712): 687-90.
	A reduced K+ current due to a novel mutation in KCNQ2 causes neonatal convulsions., Lerche H, Biervert C, Alekov AK, Schleithoff L, Lindner M, Klinger W, Bretschneider F, Mitrovic N, Jurkat-Rott K, Bode H, Lehmann-Horn F, Steinlein OK., Ann Neurol. September 1, 1999; 46 (3): 305-12.
	Surface expression and single channel properties of KCNQ2/KCNQ3, M-type K+ channels involved in epilepsy., Schwake M, Pusch M, Kharkovets T, Jentsch TJ., J Biol Chem. May 5, 2000; 275 (18): 13343-8.
	Molecular basis for differential sensitivity of KCNQ and I(Ks) channels to the cognitive enhancer XE991., Wang HS, Brown BS, McKinnon D, Cohen IS., Mol Pharmacol. June 1, 2000; 57 (6): 1218-23.
	Molecular cloning and functional expression of KCNQ5, a potassium channel subunit that may contribute to neuronal M-current diversity., Lerche C, Scherer CR, Seebohm G, Derst C, Wei AD, Busch AE, Steinmeyer K., J Biol Chem. July 21, 2000; 275 (29): 22395-400.
	Modulation of KCNQ2/3 potassium channels by the novel anticonvulsant retigabine., Main MJ, Cryan JE, Dupere JR, Cox B, Clare JJ, Burbidge SA., Mol Pharmacol. August 1, 2000; 58 (2): 253-62.
	Characterization of KCNQ5/Q3 potassium channels expressed in mammalian cells., Wickenden AD, Zou A, Wagoner PK, Jegla T., Br J Pharmacol. January 1, 2001; 132 (2): 381-4.
	Xe991 reveals differences in K(+) channels regulating chloride secretion in murine airway and colonic epithelium., MacVinish LJ, Guo Y, Dixon AK, Murrell-Lagnado RD, Cuthbert AW., Mol Pharmacol. October 1, 2001; 60 (4): 753-60.
	Myokymia and neonatal epilepsy caused by a mutation in the voltage sensor of the KCNQ2 K+ channel., Dedek K, Kunath B, Kananura C, Reuner U, Jentsch TJ, Steinlein OK., Proc Natl Acad Sci U S A. October 9, 2001; 98 (21): 12272-7.
	Benign familial neonatal convulsions caused by altered gating of KCNQ2/KCNQ3 potassium channels., Castaldo P, del Giudice EM, Coppola G, Pascotto A, Annunziato L, Taglialatela M., J Neurosci. January 15, 2002; 22 (2): RC199.
	A carboxy-terminal domain determines the subunit specificity of KCNQ K+ channel assembly., Schwake M, Jentsch TJ, Friedrich T., EMBO Rep. January 1, 2003; 4 (1): 76-81.
	C-terminal interaction of KCNQ2 and KCNQ3 K+ channels., Maljevic S, Lerche C, Seebohm G, Alekov AK, Busch AE, Lerche H., J Physiol. April 15, 2003; 548 (Pt 2): 353-60.
	KCNQ1 channels sense small changes in cell volume., Grunnet M, Jespersen T, MacAulay N, Jørgensen NK, Schmitt N, Pongs O, Olesen SP, Klaerke DA., J Physiol. June 1, 2003; 549 (Pt 2): 419-27.
	Inhibitory effects of pimozide on cloned and native voltage-gated potassium channels., Zhang ZH, Lee YT, Rhodes K, Wang K, Argentieri TM, Wang Q., Brain Res Mol Brain Res. July 4, 2003; 115 (1): 29-38.
	Mutations linked to generalized epilepsy in humans reduce GABA(A) receptor current., Macdonald RL, Bianchi MT, Bianch MT, Feng H., Exp Neurol. November 1, 2003; 184 Suppl 1 S58-67.
	The therapeutic potential of neuronal KCNQ channel modulators., Gribkoff VK., Expert Opin Ther Targets. December 1, 2003; 7 (6): 737-48.
	KCNQ2 and KCNQ3 potassium channel genes in benign familial neonatal convulsions: expansion of the functional and mutation spectrum., Singh NA, Westenskow P, Charlier C, Pappas C, Leslie J, Dillon J, Anderson VE, Sanguinetti MC, Leppert MF, BFNC Physician Consortium., Brain. December 1, 2003; 126 (Pt 12): 2726-37.
	Three mechanisms underlie KCNQ2/3 heteromeric potassium M-channel potentiation., Etxeberria A, Santana-Castro I, Regalado MP, Aivar P, Villarroel A., J Neurosci. October 13, 2004; 24 (41): 9146-52.
	Conditional transgenic suppression of M channels in mouse brain reveals functions in neuronal excitability, resonance and behavior., Peters HC, Hu H, Pongs O, Storm JF, Isbrandt D., Nat Neurosci. January 1, 2005; 8 (1): 51-60.
	Molecular determinants of KCNQ (Kv7) K+ channel sensitivity to the anticonvulsant retigabine., Schenzer A, Friedrich T, Pusch M, Saftig P, Jentsch TJ, Grötzinger J, Schwake M., J Neurosci. May 18, 2005; 25 (20): 5051-60.
	Identification by mass spectrometry and functional characterization of two phosphorylation sites of KCNQ2/KCNQ3 channels., Surti TS, Huang L, Jan YN, Jan LY, Cooper EC., Proc Natl Acad Sci U S A. December 6, 2005; 102 (49): 17828-33.
	A spontaneous mutation involving Kcnq2 (Kv7.2) reduces M-current density and spike frequency adaptation in mouse CA1 neurons., Otto JF, Yang Y, Frankel WN, White HS, Wilcox KS., J Neurosci. February 15, 2006; 26 (7): 2053-9.
	Structural determinants of M-type KCNQ (Kv7) K+ channel assembly., Schwake M, Athanasiadu D, Beimgraben C, Blanz J, Beck C, Jentsch TJ, Saftig P, Friedrich T., J Neurosci. April 5, 2006; 26 (14): 3757-66.
	Subthreshold changes of voltage-dependent activation of the K(V)7.2 channel in neonatal epilepsy., Hunter J, Maljevic S, Shankar A, Siegel A, Weissman B, Holt P, Olson L, Lerche H, Escayg A., Neurobiol Dis. October 1, 2006; 24 (1): 194-201.
	Regulation of the voltage-gated K(+) channels KCNQ2/3 and KCNQ3/5 by ubiquitination. Novel role for Nedd4-2., Ekberg J, Schuetz F, Boase NA, Conroy SJ, Manning J, Kumar S, Poronnik P, Adams DJ., J Biol Chem. April 20, 2007; 282 (16): 12135-42.
	Peripheral nerve hyperexcitability due to dominant-negative KCNQ2 mutations., Wuttke TV, Jurkat-Rott K, Paulus W, Garncarek M, Lehmann-Horn F, Lerche H., Neurology. November 27, 2007; 69 (22): 2045-53.
	Neutralization of a negative charge in the S1-S2 region of the KV7.2 (KCNQ2) channel affects voltage-dependent activation in neonatal epilepsy., Wuttke TV, Penzien J, Fauler M, Seebohm G, Lehmann-Horn F, Lerche H, Jurkat-Rott K., J Physiol. January 15, 2008; 586 (2): 545-55.
	Second coiled-coil domain of KCNQ channel controls current expression and subfamily specific heteromultimerization by salt bridge networks., Nakajo K, Kubo Y., J Physiol. June 15, 2008; 586 (12): 2827-40.
	Regulation of the voltage-gated K(+) channels KCNQ2/3 and KCNQ3/5 by serum- and glucocorticoid-regulated kinase-1., Schuetz F, Kumar S, Poronnik P, Adams DJ., Am J Physiol Cell Physiol. July 1, 2008; 295 (1): C73-80.
	A schizophrenia-linked mutation in PIP5K2A fails to activate neuronal M channels., Fedorenko O, Strutz-Seebohm N, Henrion U, Ureche ON, Lang F, Seebohm G, Lang UE., Psychopharmacology (Berl). July 1, 2008; 199 (1): 47-54.
	KCNQ2 and KCNQ3 mutations contribute to different idiopathic epilepsy syndromes., Neubauer BA, Waldegger S, Heinzinger J, Hahn A, Kurlemann G, Fiedler B, Eberhard F, Muhle H, Stephani U, Garkisch S, Eeg-Olofsson O, Müller U, Sander T., Neurology. July 15, 2008; 71 (3): 177-83.
	Refinement of the binding site and mode of action of the anticonvulsant Retigabine on KCNQ K+ channels., Lange W, Geissendörfer J, Schenzer A, Grötzinger J, Seebohm G, Friedrich T, Schwake M., Mol Pharmacol. February 1, 2009; 75 (2): 272-80.
	Coupling between the voltage-sensing and phosphatase domains of Ci-VSP., Villalba-Galea CA, Miceli F, Taglialatela M, Bezanilla F., J Gen Physiol. July 1, 2009; 134 (1): 5-14.
	Selective interaction of syntaxin 1A with KCNQ2: possible implications for specific modulation of presynaptic activity., Regev N, Degani-Katzav N, Korngreen A, Etzioni A, Siloni S, Alaimo A, Chikvashvili D, Villarroel A, Attali B, Lotan I., PLoS One. August 13, 2009; 4 (8): e6586.
	The acrylamide (S)-2 as a positive and negative modulator of Kv7 channels expressed in Xenopus laevis oocytes., Blom SM, Schmitt N, Jensen HS., PLoS One. December 11, 2009; 4 (12): e8251.
	The KCNQ5 potassium channel mediates a component of the afterhyperpolarization current in mouse hippocampus., Tzingounis AV, Heidenreich M, Kharkovets T, Spitzmaul G, Jensen HS, Nicoll RA, Jentsch TJ., Proc Natl Acad Sci U S A. June 1, 2010; 107 (22): 10232-7.
	A pore residue of the KCNQ3 potassium M-channel subunit controls surface expression., Gómez-Posada JC, Etxeberría A, Roura-Ferrer M, Areso P, Masin M, Murrell-Lagnado RD, Villarroel A., J Neurosci. July 7, 2010; 30 (27): 9316-23.
	Regulation of neuronal M-channel gating in an isoform-specific manner: functional interplay between calmodulin and syntaxin 1A., Etzioni A, Siloni S, Chikvashvilli D, Strulovich R, Sachyani D, Regev N, Greitzer-Antes D, Hirsch JA, Lotan I., J Neurosci. October 5, 2011; 31 (40): 14158-71.
	Surface expression and subunit specific control of steady protein levels by the Kv7.2 helix A-B linker., Aivar P, Fernández-Orth J, Gomis-Perez C, Alberdi A, Alaimo A, Rodríguez MS, Giraldez T, Miranda P, Areso P, Villarroel A., PLoS One. January 1, 2012; 7 (10): e47263.
	Coupling of Ci-VSP modules requires a combination of structure and electrostatics within the linker., Hobiger K, Utesch T, Mroginski MA, Friedrich T., Biophys J. March 21, 2012; 102 (6): 1313-22.
	De novo gain-of-function KCNT1 channel mutations cause malignant migrating partial seizures of infancy., Barcia G, Fleming MR, Deligniere A, Gazula VR, Brown MR, Langouet M, Chen H, Kronengold J, Abhyankar A, Cilio R, Nitschke P, Kaminska A, Boddaert N, Casanova JL, Desguerre I, Munnich A, Dulac O, Kaczmarek LK, Colleaux L, Nabbout R., Nat Genet. November 1, 2012; 44 (11): 1255-9.
	Impaired ion channel function related to a common KCNQ1 mutation - implications for risk stratification in long QT syndrome 1., Aidery P, Kisselbach J, Schweizer PA, Becker R, Katus HA, Thomas D., Gene. December 10, 2012; 511 (1): 26-33.
	The new KCNQ2 activator 4-Chlor-N-(6-chlor-pyridin-3-yl)-benzamid displays anticonvulsant potential., Boehlen A, Schwake M, Dost R, Kunert A, Fidzinski P, Heinemann U, Gebhardt C., Br J Pharmacol. March 1, 2013; 168 (5): 1182-200.
	Dysfunction of the Heteromeric KV7.3/KV7.5 Potassium Channel is Associated with Autism Spectrum Disorders., Gilling M, Rasmussen HB, Calloe K, Sequeira AF, Baretto M, Oliveira G, Almeida J, Lauritsen MB, Ullmann R, Boonen SE, Brondum-Nielsen K, Kalscheuer VM, Tümer Z, Vicente AM, Schmitt N, Tommerup N., Front Genet. April 16, 2013; 4 54.
	The linker pivot in Ci-VSP: the key to unlock catalysis., Hobiger K, Utesch T, Mroginski MA, Seebohm G, Friedrich T., PLoS One. July 1, 2013; 8 (7): e70272.
	Dominant-negative effects of KCNQ2 mutations are associated with epileptic encephalopathy., Orhan G, Bock M, Schepers D, Ilina EI, Reichel SN, Löffler H, Jezutkovic N, Weckhuysen S, Mandelstam S, Suls A, Danker T, Guenther E, Scheffer IE, De Jonghe P, Lerche H, Maljevic S., Ann Neurol. March 1, 2014; 75 (3): 382-94.
	Capturing distinct KCNQ2 channel resting states by metal ion bridges in the voltage-sensor domain., Gourgy-Hacohen O, Kornilov P, Pittel I, Peretz A, Attali B, Paas Y., J Gen Physiol. December 1, 2014; 144 (6): 513-27.
	Atomic basis for therapeutic activation of neuronal potassium channels., Kim RY, Yau MC, Galpin JD, Seebohm G, Ahern CA, Pless SA, Kurata HT., Nat Commun. September 3, 2015; 6 8116.

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