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A model for investigating developmental eye repair in Xenopus laevis. , Kha CX , Son PH , Lauper J, Tseng KA ., Exp Eye Res. April 1, 2018; 169 38-47.
no privacy, a Xenopus tropicalis mutant, is a model of human Hermansky-Pudlak Syndrome and allows visualization of internal organogenesis during tadpole development. , Nakayama T , Nakajima K , Cox A, Fisher M , Fisher M , Howell M, Fish MB, Yaoita Y , Grainger RM ., Dev Biol. June 15, 2017; 426 (2): 472-486.
Non-native R1 substitution in the s4 domain uniquely alters Kv4.3 channel gating. , Skerritt MR, Campbell DL., PLoS One. January 1, 2008; 3 (11): e3773.
Overexpressed Ca(v)beta3 inhibits N-type ( Cav2.2) calcium channel currents through a hyperpolarizing shift of ultra-slow and closed-state inactivation. , Yasuda T, Lewis RJ, Adams DJ., J Gen Physiol. April 1, 2004; 123 (4): 401-16.
Dopamine D2-receptor activation differentially inhibits N- and R-type Ca2+ channels in Xenopus melanotrope cells. , Zhang H , Jenks BG , Ciccarelli A, Roubos EW , Scheenen WJ., Neuroendocrinology. January 1, 2004; 80 (6): 368-78.
Functional heterogeneity of ROMK mutations linked to hyperprostaglandin E syndrome. , Jeck N, Derst C, Wischmeyer E, Ott H, Weber S, Rudin C, Seyberth HW, Daut J, Karschin A, Konrad M., Kidney Int. May 1, 2001; 59 (5): 1803-11.
Gating of inward-rectifier K+ channels by intracellular pH. , Schulte U, Fakler B., Eur J Biochem. October 1, 2000; 267 (19): 5837-41.
Deletion of the S3-S4 linker in the Shaker potassium channel reveals two quenching groups near the outside of S4. , Sørensen JB, Cha A, Latorre R, Rosenman E, Bezanilla F., J Gen Physiol. February 1, 2000; 115 (2): 209-22.
A hyperprostaglandin E syndrome mutation in Kir1.1 (renal outer medullary potassium) channels reveals a crucial residue for channel function in Kir1.3 channels. , Derst C, Wischmeyer E, Preisig-Müller R, Spauschus A, Konrad M, Hensen P, Jeck N, Seyberth HW, Daut J, Karschin A., J Biol Chem. September 11, 1998; 273 (37): 23884-91.
Correlation between charge movement and ionic current during slow inactivation in Shaker K+ channels. , Olcese R, Latorre R, Toro L, Bezanilla F, Stefani E., J Gen Physiol. November 1, 1997; 110 (5): 579-89.