Papers associated with kcnj5

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	N-terminal tyrosine residues within the potassium channel Kir3 modulate GTPase activity of Galphai., Ippolito DL, Temkin PA, Rogalski SL, Chavkin C., J Biol Chem. September 6, 2002; 277 (36): 32692-6.
	Molecular determinants for activation of G-protein-coupled inward rectifier K+ (GIRK) channels by extracellular acidosis., Mao J, Li L, McManus M, Wu J, Cui N, Jiang C., J Biol Chem. November 29, 2002; 277 (48): 46166-71.
	Early embryonic expression of ion channels and pumps in chick and Xenopus development., Rutenberg J, Cheng SM, Levin M., Dev Dyn. December 1, 2002; 225 (4): 469-84.
	Single channel analysis of the regulation of GIRK1/GIRK4 channels by protein phosphorylation., Müllner D., Biophys J. February 1, 2003; 84 (2 Pt 1): 1399-409.
	Inhibition of G-protein-coupled inward rectifying K+ channels by intracellular acidosis., Mao J, Wu J, Chen F, Wang X, Jiang C., J Biol Chem. February 28, 2003; 278 (9): 7091-8.
	The selectivity filter may act as the agonist-activated gate in the G protein-activated Kir3.1/Kir3.4 K+ channel., Claydon TW, Makary SY, Dibb KM, Boyett MR., J Biol Chem. December 12, 2003; 278 (50): 50654-63.
	Molecular basis for the inhibition of G protein-coupled inward rectifier K(+) channels by protein kinase C., Mao J, Wang X, Chen F, Wang R, Rojas A, Shi Y, Piao H, Jiang C., Proc Natl Acad Sci U S A. January 27, 2004; 101 (4): 1087-92.
	Mechanosensitivity of GIRK channels is mediated by protein kinase C-dependent channel-phosphatidylinositol 4,5-bisphosphate interaction., Zhang L, Lee JK, Lee JK, John SA, Uozumi N, Kodama I., J Biol Chem. February 20, 2004; 279 (8): 7037-47.
	Molecular determinants responsible for differential cellular distribution of G protein-gated inwardly rectifying K+ channels., Mirshahi T, Logothetis DE., J Biol Chem. March 19, 2004; 279 (12): 11890-7.
	Short variable sequence acquired in evolution enables selective inhibition of various inward-rectifier K+ channels., Ramu Y, Klem AM, Lu Z., Biochemistry. August 24, 2004; 43 (33): 10701-9.
	K+ activation of kir3.1/kir3.4 and kv1.4 K+ channels is regulated by extracellular charges., Claydon TW, Makary SY, Dibb KM, Boyett MR., Biophys J. October 1, 2004; 87 (4): 2407-18.
	RFamide-related peptides signal through the neuropeptide FF receptor and regulate pain-related responses in the rat., Pertovaara A, Ostergård M, Ankö ML, Lehti-Koivunen S, Brandt A, Hong W, Korpi ER, Panula P., Neuroscience. January 1, 2005; 134 (3): 1023-32.
	Mutation of critical GIRK subunit residues disrupts N- and C-termini association and channel function., Sarac R, Hou P, Hurley KM, Hriciste D, Cohen NA, Nelson DJ., J Neurosci. February 16, 2005; 25 (7): 1836-46.
	A difference in inward rectification and polyamine block and permeation between the Kir2.1 and Kir3.1/Kir3.4 K+ channels., Makary SM, Claydon TW, Enkvetchakul D, Nichols CG, Boyett MR., J Physiol. November 1, 2005; 568 (Pt 3): 749-66.
	Base of pore loop is important for rectification, activation, permeation, and block of Kir3.1/Kir3.4., Makary SM, Claydon TW, Dibb KM, Boyett MR., Biophys J. June 1, 2006; 90 (11): 4018-34.
	The GIRK1 brain variant GIRK1d and its functional impact on heteromultimeric GIRK channels., Steinecker B, Rosker C, Schreibmayer W., J Recept Signal Transduct Res. January 1, 2007; 27 (5-6): 369-82.
	Inhibition by cocaine of G protein-activated inwardly rectifying K+ channels expressed in Xenopus oocytes., Kobayashi T, Nishizawa D, Iwamura T, Ikeda K., Toxicol In Vitro. June 1, 2007; 21 (4): 656-64.
	Characterizations of a loss-of-function mutation in the Kir3.4 channel subunit., Calloe K, Ravn LS, Schmitt N, Sui JL, Duno M, Haunso S, Grunnet M, Svendsen JH, Olesen SP., Biochem Biophys Res Commun. December 28, 2007; 364 (4): 889-95.
	Subunit stoichiometry of heterologously expressed G-protein activated inwardly rectifying potassium channels analysed by fluorescence intensity ratio measurement., Grasser E, Steinecker B, Ahammer H, Schreibmayer W., Pflugers Arch. March 1, 2008; 455 (6): 1017-24.
	Specificity of Gbetagamma signaling depends on Galpha subunit coupling with G-protein-sensitive K(+) channels., Geng X, Du XN, Rusinova R, Liu BY, Li F, Zhang X, Chen XJ, Logothetis DE, Zhang HL., Pharmacology. January 1, 2009; 84 (2): 82-90.
	Heteromeric assembly of inward rectifier channel subunit Kir2.1 with Kir3.1 and with Kir3.4., Ishihara K, Yamamoto T, Kubo Y., Biochem Biophys Res Commun. March 20, 2009; 380 (4): 832-7.
	Cloning and characterisation of GIRK1 variants resulting from alternative RNA editing of the KCNJ3 gene transcript in a human breast cancer cell line., Wagner V, Stadelmeyer E, Riederer M, Regitnig P, Gorischek A, Devaney T, Schmidt K, Tritthart HA, Hirschberg K, Bauernhofer T, Schreibmayer W., J Cell Biochem. June 1, 2010; 110 (3): 598-608.
	Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants., Kobayashi T, Washiyama K, Ikeda K., PLoS One. January 1, 2011; 6 (12): e28208.
	Crystal structure of the mammalian GIRK2 K+ channel and gating regulation by G proteins, PIP2, and sodium., Whorton MR, MacKinnon R., Cell. September 30, 2011; 147 (1): 199-208.
	Characterization of a novel somatic KCNJ5 mutation delI157 in an aldosterone-producing adenoma., Murthy M, Azizan EA, Brown MJ, O'Shaughnessy KM., J Hypertens. September 1, 2012; 30 (9): 1827-33.
	Molecular basis of the facilitation of the heterooligomeric GIRK1/GIRK4 complex by cAMP dependent protein kinase., Treiber F, Rosker C, Keren-Raifman T, Steinecker B, Gorischek A, Dascal N, Schreibmayer W., Biochim Biophys Acta. April 1, 2013; 1828 (4): 1214-21.
	RGS4 regulates partial agonism of the M2 muscarinic receptor-activated K+ currents., Chen IS, Furutani K, Inanobe A, Kurachi Y., J Physiol. March 15, 2014; 592 (6): 1237-48.
	A Kir3.4 mutation causes Andersen-Tawil syndrome by an inhibitory effect on Kir2.1., Kokunai Y, Nakata T, Furuta M, Sakata S, Kimura H, Aiba T, Yoshinaga M, Osaki Y, Nakamori M, Itoh H, Sato T, Kubota T, Kadota K, Shindo K, Mochizuki H, Shimizu W, Horie M, Okamura Y, Ohno K, Takahashi MP., Neurology. March 25, 2014; 82 (12): 1058-64.
	Role for germline mutations and a rare coding single nucleotide polymorphism within the KCNJ5 potassium channel in a large cohort of sporadic cases of primary aldosteronism., Murthy M, Xu S, Massimo G, Wolley M, Gordon RD, Stowasser M, O'Shaughnessy KM., Hypertension. April 1, 2014; 63 (4): 783-9.
	Differential effects of genetically-encoded Gβγ scavengers on receptor-activated and basal Kir3.1/Kir3.4 channel current in rat atrial myocytes., Kienitz MC, Mintert-Jancke E, Hertel F, Pott L., Cell Signal. June 1, 2014; 26 (6): 1182-1192.
	Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation., Mesirca P, Alig J, Torrente AG, Müller JC, Marger L, Rollin A, Marquilly C, Vincent A, Dubel S, Bidaud I, Fernandez A, Seniuk A, Engeland B, Singh J, Miquerol L, Ehmke H, Eschenhagen T, Nargeot J, Wickman K, Isbrandt D, Mangoni ME., Nat Commun. August 21, 2014; 5 4664.
	9B.03: A NOVEL INSERTIONAL SOMATIC KCNJ5 MUTATION IN AN AUSTRALIAN PATIENT WITH AN ALDOSTERONE PRODUCING ADENOMA., Xu S, Hardege I, Murthy M, Gordon R, Stowasser M, OʼShaughnessy K., J Hypertens. June 1, 2015; 33 Suppl 1 e120.
	Identification of the Intracellular Na+ Sensor in Slo2.1 Potassium Channels., Thomson SJ, Hansen A, Sanguinetti MC., J Biol Chem. June 5, 2015; 290 (23): 14528-35.
	Novel Insertion Mutation in KCNJ5 Channel Produces Constitutive Aldosterone Release From H295R Cells., Hardege I, Xu S, Gordon RD, Thompson AJ, Figg N, Stowasser M, Murrell-Lagnado R, O'Shaughnessy KM., Mol Endocrinol. October 1, 2015; 29 (10): 1522-30.
	Ivermectin activates GIRK channels in a PIP2 -dependent, Gβγ -independent manner and an amino acid residue at the slide helix governs the activation., Chen IS, Tateyama M, Fukata Y, Uesugi M, Kubo Y., J Physiol. September 1, 2017; 595 (17): 5895-5912.
	Familial Sinus Node Disease Caused by a Gain of GIRK (G-Protein Activated Inwardly Rectifying K+ Channel) Channel Function., Kuß J, Stallmeyer B, Goldstein M, Rinné S, Pees C, Zumhagen S, Seebohm G, Decher N, Pott L, Kienitz MC, Schulze-Bahr E., Circ Genom Precis Med. January 1, 2019; 12 (1): e002238.
	Mutant KCNJ3 and KCNJ5 Potassium Channels as Novel Molecular Targets in Bradyarrhythmias and Atrial Fibrillation., Yamada N, Asano Y, Fujita M, Yamazaki S, Inanobe A, Matsuura N, Kobayashi H, Ohno S, Ebana Y, Tsukamoto O, Ishino S, Takuwa A, Kioka H, Yamashita T, Hashimoto N, Zankov DP, Shimizu A, Asakura M, Asanuma H, Kato H, Nishida Y, Miyashita Y, Shinomiya H, Naiki N, Hayashi K, Makiyama T, Ogita H, Miura K, Ueshima H, Komuro I, Yamagishi M, Horie M, Kawakami K, Furukawa T, Koizumi A, Kurachi Y, Sakata Y, Minamino T, Kitakaze M, Takashima S., Circulation. April 30, 2019; 139 (18): 2157-2169.
	The small molecule GAT1508 activates brain-specific GIRK1/2 channel heteromers and facilitates conditioned fear extinction in rodents., Xu Y, Cantwell L, Molosh AI, Plant LD, Gazgalis D, Fitz SD, Dustrude ET, Yang Y, Kawano T, Garai S, Noujaim SF, Shekhar A, Logothetis DE, Thakur GA., J Biol Chem. March 13, 2020; 295 (11): 3614-3634.
	Identification of a unique endoplasmic retention motif in the Xenopus GIRK5 channel and its contribution to oocyte maturation., Rangel-Garcia CI, Salvador C, Chavez-Garcia K, Diaz-Bello B, Lopez-Gonzalez Z, Vazquez-Cruz L, Angel Vazquez-Martinez J, Ortiz-Navarrete V, Riveros-Rosas H, Escobar LI., FEBS Open Bio. April 1, 2021; 11 (4): 1093-1108.
	Consequences of somatic mutations of GIRK1 detected in primary malign tumors on expression and function of G-protein activated, inwardly rectifying, K+ channels., Pelzmann B, Hatab A, Scheruebel S, Langthaler S, Rienmueller T, Sokolowski A, Gorischek A, Platzer D, Zorn-Pauly K, Jahn SW, Bauernhofer T, Schreibmayer W., Front Oncol. January 1, 2022; 12 998907.
	A revised mechanism of action of hyperaldosteronism-linked mutations in cytosolic domains of GIRK4 (KCNJ5)., Shalomov B, Handklo-Jamal R, Reddy HP, Theodor N, Bera AK, Dascal N., J Physiol. March 1, 2022; 600 (6): 1419-1437.

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