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XB-GENEPAGE-6042494
Papers associated with kcnt2
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Slick (Slo2.1), a rapidly-gating sodium-activated potassium channel inhibited by ATP., Bhattacharjee A, Joiner WJ, Wu M, Yang Y, Sigworth FJ, Kaczmarek LK., J Neurosci. December 17, 2003; 23 (37): 11681-91. |
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Opposite regulation of Slick and Slack K+ channels by neuromodulators., Santi CM, Ferreira G, Yang B, Gazula VR, Butler A, Wei A, Kaczmarek LK, Salkoff L., J Neurosci. May 10, 2006; 26 (19): 5059-68. |
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Amino-termini isoforms of the Slack K+ channel, regulated by alternative promoters, differentially modulate rhythmic firing and adaptation., Brown MR, Kronengold J, Gazula VR, Spilianakis CG, Flavell RA, von Hehn CA, Bhattacharjee A, Kaczmarek LK., J Physiol. November 1, 2008; 586 (21): 5161-79. |
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The N-terminal domain of Slack determines the formation and trafficking of Slick/Slack heteromeric sodium-activated potassium channels., Chen H, Kronengold J, Yan Y, Gazula VR, Brown MR, Ma L, Ferreira G, Yang Y, Bhattacharjee A, Sigworth FJ, Salkoff L, Kaczmarek LK., J Neurosci. April 29, 2009; 29 (17): 5654-65. |
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Activation of Slo2.1 channels by niflumic acid., Dai L, Garg V, Sanguinetti MC., J Gen Physiol. March 1, 2010; 135 (3): 275-95.  |
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PIP₂ modulation of Slick and Slack K⁺ channels., de los Angeles Tejada M, Jensen LJ, Klaerke DA., Biochem Biophys Res Commun. July 27, 2012; 424 (2): 208-13. |
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Structure-activity relationship of fenamates as Slo2.1 channel activators., Garg P, Sanguinetti MC., Mol Pharmacol. November 1, 2012; 82 (5): 795-802. |
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Structural basis of ion permeation gating in Slo2.1 K+ channels., Garg P, Gardner A, Garg V, Sanguinetti MC., J Gen Physiol. November 1, 2013; 142 (5): 523-42.  |
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Cell volume changes regulate slick (Slo2.1), but not slack (Slo2.2) K+ channels., Tejada MA, Stople K, Hammami Bomholtz S, Meinild AK, Poulsen AN, Klaerke DA., PLoS One. January 1, 2014; 9 (10): e110833.  |
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Intracellular ATP does not inhibit Slo2.1 K+ channels., Garg P, Sanguinetti MC., Physiol Rep. September 11, 2014; 2 (9):  |
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Identification of the Intracellular Na+ Sensor in Slo2.1 Potassium Channels., Thomson SJ, Hansen A, Sanguinetti MC., J Biol Chem. June 5, 2015; 290 (23): 14528-35. |
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Hydrophobic interactions between the S5 segment and the pore helix stabilizes the closed state of Slo2.1 potassium channels., Suzuki T, Hansen A, Sanguinetti MC., Biochim Biophys Acta. April 1, 2016; 1858 (4): 783-92. |
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Heteromeric Slick/Slack K+ channels show graded sensitivity to cell volume changes., Tejada MA, Hashem N, Calloe K, Klaerke DA., PLoS One. February 21, 2017; 12 (2): e0169914.  |
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Molecular mechanisms of Slo2 K+ channel closure., Giese MH, Gardner A, Hansen A, Sanguinetti MC., J Physiol. April 1, 2017; 595 (7): 2321-2336. |
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A De Novo Mutation in the Sodium-Activated Potassium Channel KCNT2 Alters Ion Selectivity and Causes Epileptic Encephalopathy., Gururaj S, Palmer EE, Sheehan GD, Kandula T, Macintosh R, Ying K, Morris P, Tao J, Dias KR, Zhu Y, Dinger ME, Cowley MJ, Kirk EP, Roscioli T, Sachdev R, Duffey ME, Bye A, Bhattacharjee A., Cell Rep. October 24, 2017; 21 (4): 926-933. |
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Advancements in the use of xenopus oocytes for modelling neurological disease for novel drug discovery., O'Connor EC, Kambara K, Bertrand D., Expert Opin Drug Discov. February 1, 2024; 19 (2): 173-187.  |
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