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XB-GENEPAGE-493095
Papers associated with kcnq3
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Mutations linked to generalized epilepsy in humans reduce GABA(A) receptor current., Macdonald RL, Bianchi MT, Bianch MT, Feng H., Exp Neurol. November 1, 2003; 184 Suppl 1 S58-67. |
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Inhibitory effects of pimozide on cloned and native voltage-gated potassium channels., Zhang ZH, Lee YT, Rhodes K, Wang K, Argentieri TM, Wang Q., Brain Res Mol Brain Res. July 4, 2003; 115 (1): 29-38. |
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KCNQ1 channels sense small changes in cell volume., Grunnet M, Jespersen T, MacAulay N, Jørgensen NK, Schmitt N, Pongs O, Olesen SP, Klaerke DA., J Physiol. June 1, 2003; 549 (Pt 2): 419-27. |
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C-terminal interaction of KCNQ2 and KCNQ3 K+ channels., Maljevic S, Lerche C, Seebohm G, Alekov AK, Busch AE, Lerche H., J Physiol. April 15, 2003; 548 (Pt 2): 353-60. |
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A carboxy-terminal domain determines the subunit specificity of KCNQ K+ channel assembly., Schwake M, Jentsch TJ, Friedrich T., EMBO Rep. January 1, 2003; 4 (1): 76-81. |
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Benign familial neonatal convulsions caused by altered gating of KCNQ2/KCNQ3 potassium channels., Castaldo P, del Giudice EM, Coppola G, Pascotto A, Annunziato L, Taglialatela M., J Neurosci. January 15, 2002; 22 (2): RC199. |
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Myokymia and neonatal epilepsy caused by a mutation in the voltage sensor of the KCNQ2 K+ channel., Dedek K, Kunath B, Kananura C, Reuner U, Jentsch TJ, Steinlein OK., Proc Natl Acad Sci U S A. October 9, 2001; 98 (21): 12272-7. |
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Xe991 reveals differences in K(+) channels regulating chloride secretion in murine airway and colonic epithelium., MacVinish LJ, Guo Y, Dixon AK, Murrell-Lagnado RD, Cuthbert AW., Mol Pharmacol. October 1, 2001; 60 (4): 753-60. |
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Characterization of KCNQ5/Q3 potassium channels expressed in mammalian cells., Wickenden AD, Zou A, Wagoner PK, Jegla T., Br J Pharmacol. January 1, 2001; 132 (2): 381-4. |
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Modulation of KCNQ2/3 potassium channels by the novel anticonvulsant retigabine., Main MJ, Cryan JE, Dupere JR, Cox B, Clare JJ, Burbidge SA., Mol Pharmacol. August 1, 2000; 58 (2): 253-62. |
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Molecular cloning and functional expression of KCNQ5, a potassium channel subunit that may contribute to neuronal M-current diversity., Lerche C, Scherer CR, Seebohm G, Derst C, Wei AD, Busch AE, Steinmeyer K., J Biol Chem. July 21, 2000; 275 (29): 22395-400. |
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Molecular basis for differential sensitivity of KCNQ and I(Ks) channels to the cognitive enhancer XE991., Wang HS, Brown BS, McKinnon D, Cohen IS., Mol Pharmacol. June 1, 2000; 57 (6): 1218-23. |
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Surface expression and single channel properties of KCNQ2/KCNQ3, M-type K+ channels involved in epilepsy., Schwake M, Pusch M, Kharkovets T, Jentsch TJ., J Biol Chem. May 5, 2000; 275 (18): 13343-8. |
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A reduced K+ current due to a novel mutation in KCNQ2 causes neonatal convulsions., Lerche H, Biervert C, Alekov AK, Schleithoff L, Lindner M, Klinger W, Bretschneider F, Mitrovic N, Jurkat-Rott K, Bode H, Lehmann-Horn F, Steinlein OK., Ann Neurol. September 1, 1999; 46 (3): 305-12. |
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Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy., Schroeder BC, Kubisch C, Stein V, Jentsch TJ., Nature. December 17, 1998; 396 (6712): 687-90. |
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KCNQ2 and KCNQ3 potassium channel subunits: molecular correlates of the M-channel., Wang HS, Pan Z, Shi W, Brown BS, Wymore RS, Cohen IS, Dixon JE, McKinnon D., Science. December 4, 1998; 282 (5395): 1890-3. |
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Functional expression of two KvLQT1-related potassium channels responsible for an inherited idiopathic epilepsy., Yang WP, Levesque PC, Little WA, Conder ML, Ramakrishnan P, Neubauer MG, Blanar MA., J Biol Chem. July 31, 1998; 273 (31): 19419-23. |
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