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Summary Anatomy Item Literature (4079) Expression Attributions Wiki
XB-ANAT-86

Papers associated with tail region (and mink1)

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Functional Characterization of a Spectrum of Novel Romano-Ward Syndrome KCNQ1 Variants., Rinné S., Int J Mol Sci. January 10, 2023; 24 (2):                       

Bistable, Biphasic Regulation of PP2A-B55 Accounts for the Dynamics of Mitotic Substrate Phosphorylation., Kamenz J., Curr Biol. February 22, 2021; 31 (4): 794-808.e6.                              

Agonistic and antagonistic roles for TNIK and MINK in non-canonical and canonical Wnt signalling., Mikryukov A., PLoS One. January 1, 2012; 7 (9): e43330.                

Dynamic regulation of Emi2 by Emi2-bound Cdk1/Plk1/CK1 and PP2A-B56 in meiotic arrest of Xenopus eggs., Isoda M., Dev Cell. September 13, 2011; 21 (3): 506-19.              

Acute effects of dronedarone on both components of the cardiac delayed rectifier K+ current, HERG and KvLQT1/minK potassium channels., Thomas D., Br J Pharmacol. November 1, 2003; 140 (5): 996-1002.

Clinical and electrophysiological characterization of a novel mutation (F193L) in the KCNQ1 gene associated with long QT syndrome., Yamaguchi M., Clin Sci (Lond). April 1, 2003; 104 (4): 377-82.

Inhibitory effects of volatile anesthetics on currents produced on heterologous expression of KvLQT1 and minK in Xenopus oocytes., Chen X., Vascul Pharmacol. July 1, 2002; 39 (1-2): 33-8.

MinK subdomains that mediate modulation of and association with KvLQT1., Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.          

Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.                    

Increase of the single-channel conductance of KvLQT1 potassium channels induced by the association with minK., Pusch M., Pflugers Arch. December 1, 1998; 437 (1): 172-4.

Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits., Tristani-Firouzi M., J Physiol. July 1, 1998; 510 ( Pt 1) 37-45.

Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action., Busch AE., Br J Pharmacol. January 1, 1998; 123 (1): 23-30.

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