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Expression of Xenopus snail in mesoderm and prospective neural fold ectoderm. , Essex LJ., Dev Dyn. October 1, 1993; 198 (2): 108-22.
Expression of complement components coincides with early patterning and organogenesis in Xenopus laevis. , McLin VA ., Int J Dev Biol. January 1, 2008; 52 (8): 1123-33.
A Kir6.2 mutation causing severe functional effects in vitro produces neonatal diabetes without the expected neurological complications. , Tammaro P., Diabetologia. May 1, 2008; 51 (5): 802-10.
Activation of the K(ATP) channel by Mg-nucleotide interaction with SUR1. , Proks P., J Gen Physiol. October 1, 2010; 136 (4): 389-405.
Mutations of the same conserved glutamate residue in NBD2 of the sulfonylurea receptor 1 subunit of the KATP channel can result in either hyperinsulinism or neonatal diabetes. , Männikkö R., Diabetes. June 1, 2011; 60 (6): 1813-22.
Neurally Derived Tissues in Xenopus laevis Embryos Exhibit a Consistent Bioelectrical Left- Right Asymmetry. , Pai VP ., Stem Cells Int. January 1, 2012; 2012 353491.
A universally conserved residue in the SUR1 subunit of the KATP channel is essential for translating nucleotide binding at SUR1 into channel opening. , de Wet H., J Physiol. October 15, 2012; 590 (20): 5025-36.
Purinergic receptor-induced Ca2+ signaling in the neuroepithelium of the vomeronasal organ of larval Xenopus laevis. , Dittrich K., Purinergic Signal. January 1, 2014; 10 (2): 327-36.