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Alteration of channel activities and gating by mutations of slow ISK potassium channel. , Takumi T., J Biol Chem. November 25, 1991; 266 (33): 22192-8.
Modulation by cAMP of a slowly activating potassium channel expressed in Xenopus oocytes. , Blumenthal EM., J Neurosci. January 1, 1992; 12 (1): 290-6.
Are Xenopus oocytes unique in displaying functional IsK channel heterologous expression? , Lesage F ., Recept Channels. January 1, 1993; 1 (2): 143-52.
Time dependent changes in biophysical properties of minK channels expressed in Xenopus oocytes. , Busch AE., Biochem Biophys Res Commun. December 15, 1993; 197 (2): 473-7.
The minK potassium channel exists in functional and nonfunctional forms when expressed in the plasma membrane of Xenopus oocytes. , Blumenthal EM., J Neurosci. May 1, 1994; 14 (5 Pt 2): 3097-105.
A corticosteroid-induced gene expressing an "IsK-like" K+ channel activity in Xenopus oocytes. , Attali B., Proc Natl Acad Sci U S A. June 20, 1995; 92 (13): 6092-6.
MinK potassium channels are heteromultimeric complexes. , Tai KK., J Biol Chem. January 17, 1997; 272 (3): 1654-8.
cAMP increases apical IsK channel current and K+ secretion in vestibular dark cells. , Sunose H., J Membr Biol. March 1, 1997; 156 (1): 25-35.
Mechanism of enhancement of slow delayed rectifier current by extracellular sulfhydryl modification. , Yao JA., Am J Physiol. July 1, 1997; 273 (1 Pt 2): H208-19.
KVLQT channels are inhibited by the K+ channel blocker 293B. , Bleich M., Pflugers Arch. August 1, 1997; 434 (4): 499-501.
Suppression of slow delayed rectifier current by a truncated isoform of KvLQT1 cloned from normal human heart. , Jiang M., J Biol Chem. September 26, 1997; 272 (39): 24109-12.
Activation and inactivation of homomeric KvLQT1 potassium channels. , Pusch M., Biophys J. August 1, 1998; 75 (2): 785-92.
Single-channel properties of IKs potassium channels. , Yang Y ., J Gen Physiol. December 1, 1998; 112 (6): 665-78.
Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome. , Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
Long QT syndrome-associated mutations in the S4-S5 linker of KvLQT1 potassium channels modify gating and interaction with minK subunits. , Franqueza L., J Biol Chem. July 23, 1999; 274 (30): 21063-70.
MinK subdomains that mediate modulation of and association with KvLQT1. , Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.
minK-related peptide 1 associates with Kv4.2 and modulates its gating function: potential role as beta subunit of cardiac transient outward channel? , Zhang M., Circ Res. May 25, 2001; 88 (10): 1012-9.
RNA interference reveals that endogenous Xenopus MinK-related peptides govern mammalian K+ channel function in oocyte expression studies. , Anantharam A., J Biol Chem. April 4, 2003; 278 (14): 11739-45.
Characterization of a novel Long QT syndrome mutation G52R- KCNE1 in a Chinese family. , Ma L., Cardiovasc Res. September 1, 2003; 59 (3): 612-9.
Automated higher-throughput compound screening on ion channel targets based on the Xenopus laevis oocyte expression system. , Pehl U., Assay Drug Dev Technol. October 1, 2004; 2 (5): 515-24.
Identification and characterisation of a novel KCNQ1 mutation in a family with Romano-Ward syndrome. , Zehelein J., Biochim Biophys Acta. November 5, 2004; 1690 (3): 185-92.
KCNE3 truncation mutants reveal a bipartite modulation of KCNQ1 K+ channels. , Gage SD., J Gen Physiol. December 1, 2004; 124 (6): 759-71.
Endogenous KCNE subunits govern Kv2.1 K+ channel activation kinetics in Xenopus oocyte studies. , Gordon E., Biophys J. February 15, 2006; 90 (4): 1223-31.
Secondary structure of a KCNE cytoplasmic domain. , Rocheleau JM., J Gen Physiol. December 1, 2006; 128 (6): 721-9.
KCNE1 and KCNE3 stabilize and/or slow voltage sensing S4 segment of KCNQ1 channel. , Nakajo K., J Gen Physiol. September 1, 2007; 130 (3): 269-81.
The KCNE1 beta-subunit exerts a transient effect on the KCNQ1 K+ channel. , Poulsen AN., Biochem Biophys Res Commun. November 9, 2007; 363 (1): 133-9.
KCNQ1 and KCNE1 in the IKs channel complex make state-dependent contacts in their extracellular domains. , Xu X., J Gen Physiol. June 1, 2008; 131 (6): 589-603.
Modulation of potassium channel function confers a hyperproliferative invasive phenotype on embryonic stem cells. , Morokuma J., Proc Natl Acad Sci U S A. October 28, 2008; 105 (43): 16608-13.
The G314S KCNQ1 mutation exerts a dominant-negative effect on expression of KCNQ1 channels in oocytes. , Li W ., Biochem Biophys Res Commun. May 29, 2009; 383 (2): 206-9.
Functional delivery of a membrane protein into oocyte membranes using bicelles. , Kang C., Biochemistry. February 2, 2010; 49 (4): 653-5.
Biophysical properties of mutant KCNQ1 S277L channels linked to hereditary long QT syndrome with phenotypic variability. , Aidery P., Biochim Biophys Acta. April 1, 2011; 1812 (4): 488-94.
KCNQ1 subdomains involved in KCNE modulation revealed by an invertebrate KCNQ1 orthologue. , Nakajo K., J Gen Physiol. November 1, 2011; 138 (5): 521-35.
Inhibition of cardiac Kir2.1-2.3 channels by beta3 adrenoreceptor antagonist SR 59230A. , Kulzer M., Biochem Biophys Res Commun. July 27, 2012; 424 (2): 315-20.
AMP-activated protein kinase in BK-channel regulation and protection against hearing loss following acoustic overstimulation. , Föller M., FASEB J. October 1, 2012; 26 (10): 4243-53.
Impaired ion channel function related to a common KCNQ1 mutation - implications for risk stratification in long QT syndrome 1. , Aidery P., Gene. December 10, 2012; 511 (1): 26-33.
Domain-domain interactions determine the gating, permeation, pharmacology, and subunit modulation of the IKs ion channel. , Zaydman MA., Elife. March 12, 2014; 3 e03606.
Functional assembly of Kv7.1/ Kv7.5 channels with emerging properties on vascular muscle physiology. , Oliveras A., Arterioscler Thromb Vasc Biol. July 1, 2014; 34 (7): 1522-30.
Ginseng gintonin activates the human cardiac delayed rectifier K+ channel: involvement of Ca2+/calmodulin binding sites. , Choi SH., Mol Cells. September 1, 2014; 37 (9): 656-63.
KCNE1 induces fenestration in the Kv7.1/ KCNE1 channel complex that allows for highly specific pharmacological targeting. , Wrobel E., Nat Commun. October 12, 2016; 7 12795.
Insulin treatment augments KCNQ1/ KCNE1 currents but not KCNQ1 currents, which is associated with an increase in KCNE1 expression. , Wu M., Biochem Biophys Res Commun. November 4, 2017; 493 (1): 409-415.
Direct neurotransmitter activation of voltage-gated potassium channels. , Manville RW., Nat Commun. May 10, 2018; 9 (1): 1847.
KCNE1 tunes the sensitivity of KV7.1 to polyunsaturated fatty acids by moving turret residues close to the binding site. , Larsson JE., Elife. July 17, 2018; 7
Nocturnal Atrial Fibrillation Caused by Mutation in KCND2, Encoding Pore-Forming (α) Subunit of the Cardiac Kv4.2 Potassium Channel. , Drabkin M., Circ Genom Precis Med. November 1, 2018; 11 (11): e002293.