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Inverse agonist abolishes desensitization of a constitutively active mutant of thyrotropin-releasing hormone receptor: role of cellular calcium and protein kinase C. , Grimberg H., Br J Pharmacol. March 1, 1999; 126 (5): 1097-106.
Truncation of the thyrotropin-releasing hormone receptor carboxyl tail causes constitutive activity and leads to impaired responsiveness in Xenopus oocytes and AtT20 cells. , Matus-Leibovitch N., J Biol Chem. January 20, 1995; 270 (3): 1041-7.
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Independent external calcium entry and cellular calcium mobilization in Xenopus oocytes. , Lupu-Meiri M., Cell Calcium. July 1, 1994; 16 (1): 20-8.
Differential effects of cytoskeletal agents on hemispheric functional expression of cell membrane receptors in Xenopus oocytes. , Matus-Leibovitch N., Cell Mol Neurobiol. December 1, 1993; 13 (6): 625-37.
Molecular cloning of a functional human thyrotropin-releasing hormone receptor. , Matre V., Biochem Biophys Res Commun. August 31, 1993; 195 (1): 179-85.
G alpha 11 and G alpha q guanine nucleotide regulatory proteins differentially modulate the response to thyrotropin-releasing hormone in Xenopus oocytes. , Lipinsky D., FEBS Lett. July 28, 1992; 307 (2): 237-40.
Thyrotropin-releasing hormone ( TRH) and phorbol myristate acetate decrease TRH receptor messenger RNA in rat pituitary GH3 cells: evidence that protein kinase-C mediates the TRH effect. , Fujimoto J., Mol Endocrinol. October 1, 1991; 5 (10): 1527-32.
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