Papers associated with left (and kcnma1)

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	Gating properties conferred on BK channels by the beta3b auxiliary subunit in the absence of its NH(2)- and COOH termini., Zeng XH., J Gen Physiol. June 1, 2001; 117 (6): 607-28.
	Inactivation of BK channels mediated by the NH(2) terminus of the beta3b auxiliary subunit involves a two-step mechanism: possible separation of binding and blockade., Lingle CJ., J Gen Physiol. June 1, 2001; 117 (6): 583-606.
	Allosteric regulation of BK channel gating by Ca(2+) and Mg(2+) through a nonselective, low affinity divalent cation site., Zhang X., J Gen Physiol. November 1, 2001; 118 (5): 607-36.
	Inactivation of BK channels by the NH2 terminus of the beta2 auxiliary subunit: an essential role of a terminal peptide segment of three hydrophobic residues., Xia XM., J Gen Physiol. February 1, 2003; 121 (2): 125-48.
	Regulation of K+ flow by a ring of negative charges in the outer pore of BKCa channels. Part II: Neutralization of aspartate 292 reduces long channel openings and gating current slow component., Haug T., J Gen Physiol. August 1, 2004; 124 (2): 185-97.
	Regulation of K+ flow by a ring of negative charges in the outer pore of BKCa channels. Part I: Aspartate 292 modulates K+ conduction by external surface charge effect., Haug T., J Gen Physiol. August 1, 2004; 124 (2): 173-84.
	Slo3 K+ channels: voltage and pH dependence of macroscopic currents., Zhang X., J Gen Physiol. September 1, 2006; 128 (3): 317-36.
	pH-regulated Slo3 K+ channels: properties of unitary currents., Zhang X., J Gen Physiol. September 1, 2006; 128 (3): 301-15.
	Species-specific Differences among KCNMB3 BK beta3 auxiliary subunits: some beta3 N-terminal variants may be primate-specific subunits., Zeng X., J Gen Physiol. July 1, 2008; 132 (1): 115-29.
	Interactions between beta subunits of the KCNMB family and Slo3: beta4 selectively modulates Slo3 expression and function., Yang CT., PLoS One. July 3, 2009; 4 (7): e6135.
	Fast and slow gating are inherent properties of the pore module of the K+ channel Kcv., Abenavoli A., J Gen Physiol. September 1, 2009; 134 (3): 219-29.
	Closed-channel block of BK potassium channels by bbTBA requires partial activation., Tang QY., J Gen Physiol. November 1, 2009; 134 (5): 409-36.
	Block of mouse Slo1 and Slo3 K+ channels by CTX, IbTX, TEA, 4-AP and quinidine., Tang QY., Channels (Austin). January 1, 2010; 4 (1): 22-41.
	Activation of Slo2.1 channels by niflumic acid., Dai L., J Gen Physiol. March 1, 2010; 135 (3): 275-95.
	Glycine311, a determinant of paxilline block in BK channels: a novel bend in the BK S6 helix., Zhou Y., J Gen Physiol. May 1, 2010; 135 (5): 481-94.
	Histone variant macroH2A confers resistance to nuclear reprogramming., Pasque V., EMBO J. May 6, 2011; 30 (12): 2373-87.
	Both transmembrane domains of BK β1 subunits are essential to confer the normal phenotype of β1-containing BK channels., Kuntamallappanavar G., PLoS One. January 1, 2014; 9 (10): e109306.
	Paxilline inhibits BK channels by an almost exclusively closed-channel block mechanism., Zhou Y., J Gen Physiol. November 1, 2014; 144 (5): 415-40.
	Alcohol modulation of BK channel gating depends on β subunit composition., Kuntamallappanavar G., J Gen Physiol. November 1, 2016; 148 (5): 419-440.
	Role of remodeling and spacing factor 1 in histone H2A ubiquitination-mediated gene silencing., Zhang Z, Zhang Z., Proc Natl Acad Sci U S A. September 19, 2017; 114 (38): E7949-E7958.
	Novel description of the large conductance Ca2+-modulated K+ channel current, BK, during an action potential from suprachiasmatic nucleus neurons., Clay JR., Physiol Rep. November 1, 2017; 5 (20):
	Asymmetric development of the nervous system., Alqadah A., Dev Dyn. January 1, 2018; 247 (1): 124-137.
	Pituitary cell translation and secretory capacities are enhanced cell autonomously by the transcription factor Creb3l2., Khetchoumian K., Nat Commun. September 3, 2019; 10 (1): 3960.
	Advancements in the use of xenopus oocytes for modelling neurological disease for novel drug discovery., O'Connor EC., Expert Opin Drug Discov. February 1, 2024; 19 (2): 173-187.

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