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Primary structure and functional expression of a rat G-protein-coupled muscarinic potassium channel. , Kubo Y., Nature. August 26, 1993; 364 (6440): 802-6.
Cloning and functional expression of a cardiac inward rectifier K+ channel. , Ishii K., FEBS Lett. January 24, 1994; 338 (1): 107-11.
Cloning a novel human brain inward rectifier potassium channel and its functional expression in Xenopus oocytes. , Tang W., FEBS Lett. July 18, 1994; 348 (3): 239-43.
Molecular cloning and functional expression of cDNA encoding a second class of inward rectifier potassium channels in the mouse brain. , Takahashi N., J Biol Chem. September 16, 1994; 269 (37): 23274-9.
Gating mechanism of the cloned inward rectifier potassium channel from mouse heart. , Ishihara K., J Membr Biol. October 1, 1994; 142 (1): 55-64.
Molecular cloning and expression of a human heart inward rectifier potassium channel. , Raab-Graham KF., Neuroreport. December 20, 1994; 5 (18): 2501-5.
Cloning, localization, and functional expression of a human brain inward rectifier potassium channel (hIRK1). , Tang W., Recept Channels. January 1, 1995; 3 (3): 175-83.
Cloning and functional expression of an inwardly rectifying K+ channel from human atrium. , Wible BA., Circ Res. March 1, 1995; 76 (3): 343-50.
Cloning and functional characterization of a novel ATP-sensitive potassium channel ubiquitously expressed in rat tissues, including pancreatic islets, pituitary, skeletal muscle, and heart. , Inagaki N., J Biol Chem. March 17, 1995; 270 (11): 5691-4.
Susceptibility of cloned K+ channels to reactive oxygen species. , Duprat F., Proc Natl Acad Sci U S A. December 5, 1995; 92 (25): 11796-800.
Molecular and functional heterogeneity of inward rectifier potassium channels in brain and heart. , Kurachi Y., J Card Fail. December 1, 1996; 2 (4 Suppl): S59-62.
Molecular cloning and expression of a bovine endothelial inward rectifier potassium channel. , Forsyth SE., FEBS Lett. June 9, 1997; 409 (2): 277-82.
Molecular characterization of an inwardly rectifying K+ channel from HeLa cells. , Klein H., J Membr Biol. January 1, 1999; 167 (1): 43-52.
Suppression of Kir2.3 activity by protein kinase C phosphorylation of the channel protein at threonine 53. , Zhu G., J Biol Chem. April 23, 1999; 274 (17): 11643-6.
Block of inwardly rectifying K+ currents by extracellular Mg2+ and Ba2+ in bovine pulmonary artery endothelial cells. , Leung YM., Can J Physiol Pharmacol. September 1, 2000; 78 (9): 751-6.
Mutations in Kir2.1 cause the developmental and episodic electrical phenotypes of Andersen's syndrome. , Plaster NM., Cell. May 18, 2001; 105 (4): 511-9.
Identification of a site involved in the block by extracellular Mg(2+) and Ba(2+) as well as permeation of K(+) in the Kir2.1 K(+) channel. , Murata Y., J Physiol. November 1, 2002; 544 (3): 665-77.
Inhibition of G protein-activated inwardly rectifying K+ channels by fluoxetine (Prozac). , Kobayashi T., Br J Pharmacol. March 1, 2003; 138 (6): 1119-28.
Polymorphic ventricular tachycardia and KCNJ2 mutations. , Chun TU., Heart Rhythm. July 1, 2004; 1 (2): 235-41.
Functional and clinical characterization of a mutation in KCNJ2 associated with Andersen-Tawil syndrome. , Lu CW., J Med Genet. August 1, 2006; 43 (8): 653-9.
Inhibition by cocaine of G protein-activated inwardly rectifying K+ channels expressed in Xenopus oocytes. , Kobayashi T., Toxicol In Vitro. June 1, 2007; 21 (4): 656-64.
Activation of inwardly rectifying Kir2.x potassium channels by beta 3-adrenoceptors is mediated via different signaling pathways with a predominant role of PKC for Kir2.1 and of PKA for Kir2.2. , Scherer D., Naunyn Schmiedebergs Arch Pharmacol. July 1, 2007; 375 (5): 311-22.
Pregnenolone sulfate potentiates the inwardly rectifying K channel Kir2.3. , Kobayashi T., PLoS One. July 21, 2009; 4 (7): e6311.
Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants. , Kobayashi T., PLoS One. January 1, 2011; 6 (12): e28208.
A Kir3.4 mutation causes Andersen-Tawil syndrome by an inhibitory effect on Kir2.1. , Kokunai Y., Neurology. March 25, 2014; 82 (12): 1058-64.
Genetically induced dysfunctions of Kir2.1 channels: implications for short QT3 syndrome and autism-epilepsy phenotype. , Ambrosini E., Hum Mol Genet. September 15, 2014; 23 (18): 4875-86.
Class III antiarrhythmic drug dronedarone inhibits cardiac inwardly rectifying Kir2.1 channels through binding at residue E224. , Xynogalos P., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2014; 387 (12): 1153-61.
Dual Mechanism for Inhibition of Inwardly Rectifying Kir2.x Channels by Quinidine Involving Direct Pore Block and PIP2-interference. , Koepple C., J Pharmacol Exp Ther. May 1, 2017; 361 (2): 209-218.
Bioelectric signaling: Reprogrammable circuits underlying embryogenesis, regeneration, and cancer. , Levin M ., Cell. April 15, 2021;