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Regulation of nuclear envelope precursor functions during cell division. , Vigers GP., J Cell Sci. June 1, 1992; 102 ( Pt 2) 273-84.
Microtubule nucleating activity of centrosomes in cell-free extracts from Xenopus eggs: involvement of phosphorylation and accumulation of pericentriolar material. , Ohta K., J Cell Sci. January 1, 1993; 104 ( Pt 1) 125-37.
Cip1 blocks the initiation of DNA replication in Xenopus extracts by inhibition of cyclin-dependent kinases. , Strausfeld UP., Curr Biol. October 1, 1994; 4 (10): 876-83.
Control of the Cdc2/ cyclin B complex in Xenopus egg extracts arrested at a G2/M checkpoint with DNA synthesis inhibitors. , Kumagai A ., Mol Biol Cell. February 1, 1995; 6 (2): 199-213.
Activation of the Xenopus cyclin degradation machinery by full-length cyclin A. , Jones C ., J Cell Sci. May 1, 1996; 109 ( Pt 5) 1071-9.
Identification and characterization of SA/Scc3p subunits in the Xenopus and human cohesin complexes. , Losada A., J Cell Biol. August 7, 2000; 150 (3): 405-16.
The checkpoint protein Chfr is a ligase that ubiquitinates Plk1 and inhibits Cdc2 at the G2 to M transition. , Kang D., J Cell Biol. January 21, 2002; 156 (2): 249-59.
Differences in regulation of the first two M-phases in Xenopus laevis embryo cell-free extracts. , Chesnel F., Dev Biol. September 15, 2005; 285 (2): 358-75.
Cyclin B dissociation from CDK1 precedes its degradation upon MPF inactivation in mitotic extracts of Xenopus laevis embryos. , Chesnel F., Cell Cycle. August 1, 2006; 5 (15): 1687-98.
Transient activation of calcineurin is essential to initiate embryonic development in Xenopus laevis. , Nishiyama T., Nature. September 20, 2007; 449 (7160): 341-5.
Temporal regulation of embryonic M-phases. , Kubiak JZ ., Folia Histochem Cytobiol. January 1, 2008; 46 (1): 5-9.
Canonical and Alternative Pathways in Cyclin-Dependent Kinase 1/ Cyclin B Inactivation upon M-Phase Exit in Xenopus laevis Cell-Free Extracts. , Kubiak JZ ., Enzyme Res. January 1, 2011; 2011 523420.
Control of timing of embryonic M-phase entry and exit is differentially sensitive to CDK1 and PP2A balance. , El Dika M., Int J Dev Biol. January 1, 2014; 58 (10-12): 767-74.