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Summary Anatomy Item Literature (1230) Expression Attributions Wiki
XB-ANAT-3515

Papers associated with myocyte (and hcn4)

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Disease-associated HCN4 V759I variant is not sufficient to impair cardiac pacemaking., Erlenhardt N., Pflugers Arch. December 1, 2020; 472 (12): 1733-1742.      

Id genes are essential for early heart formation., Cunningham TJ., Genes Dev. July 1, 2017; 31 (13): 1325-1338.                

Direct nkx2-5 transcriptional repression of isl1 controls cardiomyocyte subtype identity., Dorn T., Stem Cells. April 1, 2015; 33 (4): 1113-29.              

Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation., Mesirca P., Nat Commun. August 21, 2014; 5 4664.                

The cAMP-binding Popdc proteins have a redundant function in the heart., Brand T., Biochem Soc Trans. April 1, 2014; 42 (2): 295-301.      

Association with the auxiliary subunit PEX5R/Trip8b controls responsiveness of HCN channels to cAMP and adrenergic stimulation., Zolles G., Neuron. June 25, 2009; 62 (6): 814-25.

Associated changes in HCN2 and HCN4 transcripts and I(f) pacemaker current in myocytes., Zhang Q., Biochim Biophys Acta. May 1, 2009; 1788 (5): 1138-47.

Shox2 is essential for the differentiation of cardiac pacemaker cells by repressing Nkx2-5., Espinoza-Lewis RA., Dev Biol. March 15, 2009; 327 (2): 376-85.      

Tyrosine kinase inhibition differentially regulates heterologously expressed HCN channels., Yu HG., Pflugers Arch. January 1, 2004; 447 (4): 392-400.

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