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Summary Anatomy Item Literature (2432) Expression Attributions Wiki
XB-ANAT-63

Papers associated with heart (and mespb)

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Membrane potential drives the exit from pluripotency and cell fate commitment via calcium and mTOR., Sempou E., Nat Commun. November 5, 2022; 13 (1): 6681.                                            


Genome-wide transcriptomics analysis identifies sox7 and sox18 as specifically regulated by gata4 in cardiomyogenesis., Afouda BA., Dev Biol. February 1, 2018; 434 (1): 108-120.                  


Id genes are essential for early heart formation., Cunningham TJ., Genes Dev. July 1, 2017; 31 (13): 1325-1338.                


E2a is necessary for Smad2/3-dependent transcription and the direct repression of lefty during gastrulation., Wills AE., Dev Cell. February 9, 2015; 32 (3): 345-57.                  


Hhex and Cer1 mediate the Sox17 pathway for cardiac mesoderm formation in embryonic stem cells., Liu Y., Stem Cells. June 1, 2014; 32 (6): 1515-26.              


In vivo T-box transcription factor profiling reveals joint regulation of embryonic neuromesodermal bipotency., Gentsch GE., Cell Rep. September 26, 2013; 4 (6): 1185-96.                              


Cardiac differentiation in Xenopus is initiated by mespa., Kriegmair MC., Cardiovasc Res. March 1, 2013; 97 (3): 454-63.


Myogenic waves and myogenic programs during Xenopus embryonic myogenesis., Della Gaspera B., Dev Dyn. May 1, 2012; 241 (5): 995-1007.                                    


Early activation of FGF and nodal pathways mediates cardiac specification independently of Wnt/beta-catenin signaling., Samuel LJ., PLoS One. October 28, 2009; 4 (10): e7650.                


The Xenopus Bowline/Ripply family proteins negatively regulate the transcriptional activity of T-box transcription factors., Hitachi K., Int J Dev Biol. January 1, 2009; 53 (4): 631-9.                    


MesP1 drives vertebrate cardiovascular differentiation through Dkk-1-mediated blockade of Wnt-signalling., David R., Nat Cell Biol. March 1, 2008; 10 (3): 338-45.

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