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Summary Anatomy Item Literature (46) Expression Attributions Wiki
XB-ANAT-1563

Papers associated with migratory neural crest cell (and twist1)

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The H2A.Z and NuRD associated protein HMG20A controls early head and heart developmental transcription programs., Herchenröther A., Nat Commun. January 28, 2023; 14 (1): 472.                                                    


Kindlin2 regulates neural crest specification via integrin-independent regulation of the FGF signaling pathway., Wang H., Development. May 15, 2021; 148 (10):                                           


Dynamic expression of MMP28 during cranial morphogenesis., Gouignard N., Philos Trans R Soc Lond B Biol Sci. October 12, 2020; 375 (1809): 20190559.


Serine Threonine Kinase Receptor-Associated Protein Deficiency Impairs Mouse Embryonic Stem Cells Lineage Commitment Through CYP26A1-Mediated Retinoic Acid Homeostasis., Jin L., Stem Cells. September 1, 2018; 36 (9): 1368-1379.                      


Redistribution of Adhesive Forces through Src/FAK Drives Contact Inhibition of Locomotion in Neural Crest., Roycroft A., Dev Cell. June 4, 2018; 45 (5): 565-579.e3.                                        


Apolipoprotein C-I mediates Wnt/Ctnnb1 signaling during neural border formation and is required for neural crest development., Yokota C., Int J Dev Biol. January 1, 2017; 61 (6-7): 415-425.                      


Controlled levels of canonical Wnt signaling are required for neural crest migration., Maj E., Dev Biol. September 1, 2016; 417 (1): 77-90.                          


Delamination of neural crest cells requires transient and reversible Wnt inhibition mediated by Dact1/2., Rabadán MA., Development. June 15, 2016; 143 (12): 2194-205.          


Cadherin Switch during EMT in Neural Crest Cells Leads to Contact Inhibition of Locomotion via Repolarization of Forces., Scarpa E., Dev Cell. August 24, 2015; 34 (4): 421-34.                                            


GSK3 and Polo-like kinase regulate ADAM13 function during cranial neural crest cell migration., Abbruzzese G., Mol Biol Cell. December 15, 2014; 25 (25): 4072-82.                                    


The LIM adaptor protein LMO4 is an essential regulator of neural crest development., Ochoa SD., Dev Biol. January 15, 2012; 361 (2): 313-25.              


Targeted inactivation of Snail family EMT regulatory factors by a Co(III)-Ebox conjugate., Harney AS., PLoS One. January 1, 2012; 7 (2): e32318.            


CHD7 cooperates with PBAF to control multipotent neural crest formation., Bajpai R., Nature. February 18, 2010; 463 (7283): 958-62.      


Mechanisms driving neural crest induction and migration in the zebrafish and Xenopus laevis., Klymkowsky MW., Cell Adh Migr. January 1, 2010; 4 (4): 595-608.  


Samba, a Xenopus hnRNP expressed in neural and neural crest tissues., Yan CY., Dev Dyn. January 1, 2009; 238 (1): 204-9.      


A Myc-Slug (Snail2)/Twist regulatory circuit directs vascular development., Rodrigues CO., Development. June 1, 2008; 135 (11): 1903-11.              


Xenopus hairy2 functions in neural crest formation by maintaining cells in a mitotic and undifferentiated state., Nagatomo K., Dev Dyn. June 1, 2007; 236 (6): 1475-83.          


Slug stability is dynamically regulated during neural crest development by the F-box protein Ppa., Vernon AE., Development. September 1, 2006; 133 (17): 3359-70.                


Xenopus Id3 is required downstream of Myc for the formation of multipotent neural crest progenitor cells., Light W., Development. April 1, 2005; 132 (8): 1831-41.              


The protooncogene c-myc is an essential regulator of neural crest formation in xenopus., Bellmeyer A., Dev Cell. June 1, 2003; 4 (6): 827-39.        


Snail-related transcriptional repressors are required in Xenopus for both the induction of the neural crest and its subsequent migration., LaBonne C., Dev Biol. May 1, 2000; 221 (1): 195-205.          


Xenopus brain factor-2 controls mesoderm, forebrain and neural crest development., Gómez-Skarmeta JL., Mech Dev. January 1, 1999; 80 (1): 15-27.              

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