Papers associated with cardiovascular system (and cav1)

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	Membrane cholesterol regulates TRPV4 function, cytoskeletal expression, and the cellular response to tension., Lakk M., J Lipid Res. January 1, 2021; 62 100145.
	Caveolin 1 is required for axonal outgrowth of motor neurons and affects Xenopus neuromuscular development., Breuer M., Sci Rep. October 5, 2020; 10 (1): 16446.
	Innate Immune Response and Off-Target Mis-splicing Are Common Morpholino-Induced Side Effects in Xenopus., Gentsch GE., Dev Cell. March 12, 2018; 44 (5): 597-610.e10.
	Protein kinase C enhances plasma membrane expression of cardiac L-type calcium channel, CaV1.2., Raifman TK., Channels (Austin). November 2, 2017; 11 (6): 604-615.
	A Mutation in the G-Protein Gene GNB2 Causes Familial Sinus Node and Atrioventricular Conduction Dysfunction., Stallmeyer B., Circ Res. May 12, 2017; 120 (10): e33-e44.
	Differential zinc permeation and blockade of L-type Ca2+ channel isoforms Cav1.2 and Cav1.3., Park SJ., Biochim Biophys Acta. October 1, 2015; 1848 (10 Pt A): 2092-100.
	Asn-Linked Glycosylation Contributes to Surface Expression and Voltage-Dependent Gating of Cav1.2 Ca²⁺ Channel., Park HJ., J Microbiol Biotechnol. August 1, 2015; 25 (8): 1371-9.
	Molecular cloning and characterization of a hamster Cav1.3 Ca2+ channel variant with a long carboxyl terminus., Kang HW., Biochim Biophys Acta. June 1, 2011; 1808 (6): 1629-38.
	Cardiac L-type calcium channel (Cav1.2) associates with gamma subunits., Yang L., FASEB J. March 1, 2011; 25 (3): 928-36.
	Cloning and characterization of voltage-gated calcium channel alpha1 subunits in Xenopus laevis during development., Lewis BB., Dev Dyn. November 1, 2009; 238 (11): 2891-902.
	Disruption of the IS6-AID linker affects voltage-gated calcium channel inactivation and facilitation., Findeisen F., J Gen Physiol. March 1, 2009; 133 (3): 327-43.
	Characterization of the calmodulin-binding site in the N terminus of CaV1.2., Benmocha A., Channels (Austin). January 1, 2009; 3 (5): 337-42.
	Regulation of maximal open probability is a separable function of Ca(v)beta subunit in L-type Ca2+ channel, dependent on NH2 terminus of alpha1C (Ca(v)1.2alpha)., Kanevsky N., J Gen Physiol. July 1, 2006; 128 (1): 15-36.
	Effects of congenital stationary night blindness type 2 mutations R508Q and L1364H on Cav1.4 L-type Ca2+ channel function and expression., Hoda JC., J Neurochem. March 1, 2006; 96 (6): 1648-58.
	Functional architecture of the inner pore of a voltage-gated Ca2+ channel., Zhen XG., J Gen Physiol. September 1, 2005; 126 (3): 193-204.
	Novel molecular mechanism involving alpha1D (Cav1.3) L-type calcium channel in autoimmune-associated sinus bradycardia., Qu Y., Circulation. June 14, 2005; 111 (23): 3034-41.
	Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations., Splawski I., Proc Natl Acad Sci U S A. June 7, 2005; 102 (23): 8089-96; discussion 8086-8.
	Roles of molecular regions in determining differences between voltage dependence of activation of CaV3.1 and CaV1.2 calcium channels., Li J., J Biol Chem. June 25, 2004; 279 (26): 26858-67.
	Overexpressed Ca(v)beta3 inhibits N-type (Cav2.2) calcium channel currents through a hyperpolarizing shift of ultra-slow and closed-state inactivation., Yasuda T., J Gen Physiol. April 1, 2004; 123 (4): 401-16.

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