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Retinoic acid-induced expression of Hnf1b and Fzd4 is required for pancreas development in Xenopus laevis. , Gere-Becker MB., Development. June 8, 2018; 145 (12):
A gene regulatory program controlling early Xenopus mesendoderm formation: Network conservation and motifs. , Charney RM ., Semin Cell Dev Biol. June 1, 2017; 66 12-24.
Xenopus as a model system for studying pancreatic development and diabetes. , Kofent J., Semin Cell Dev Biol. March 1, 2016; 51 106-16.
Recessive mutations in PCBD1 cause a new type of early-onset diabetes. , Simaite D., Diabetes. October 1, 2014; 63 (10): 3557-64.
Heat-shock mediated overexpression of HNF1β mutations has differential effects on gene expression in the Xenopus pronephric kidney. , Sauert K., PLoS One. January 1, 2012; 7 (3): e33522.
Xenopus as a model system for the study of GOLPH2/ GP73 function: Xenopus GOLPH2 is required for pronephros development. , Li L., PLoS One. January 1, 2012; 7 (6): e38939.
A heterozygous activating mutation in the sulphonylurea receptor SUR1 ( ABCC8) causes neonatal diabetes. , Proks P., Hum Mol Genet. June 1, 2006; 15 (11): 1793-800.
An alternative, human SRC promoter and its regulation by hepatic nuclear factor-1alpha. , Bonham K., J Biol Chem. December 1, 2000; 275 (48): 37604-11.
Cloning and developmental expression of LFB3/ HNF1 beta transcription factor in Xenopus laevis. , Demartis A., Mech Dev. July 1, 1994; 47 (1): 19-28.