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Summary Anatomy Item Literature (1033) Expression Attributions Wiki
XB-ANAT-3344

Papers associated with hand (and kcnj2)

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Bioelectric signalling via potassium channels: a mechanism for craniofacial dysmorphogenesis in KCNJ2-associated Andersen-Tawil Syndrome., Adams DS., J Physiol. June 15, 2016; 594 (12): 3245-70.                              

Gating of the kir2.1 channel at the bundle crossing region by intracellular spermine and other cations., Huang CW., J Cell Physiol. November 1, 2014; 229 (11): 1703-21.

The bundle crossing region is responsible for the inwardly rectifying internal spermine block of the Kir2.1 channel., Huang CW., Pflugers Arch. February 1, 2014; 466 (2): 275-93.

H bonding at the helix-bundle crossing controls gating in Kir potassium channels., Rapedius M., Neuron. August 16, 2007; 55 (4): 602-14.                

Selective inhibition of Kir currents by antihistamines., Liu B., Eur J Pharmacol. March 8, 2007; 558 (1-3): 21-6.

Mechanism of rectification in inward-rectifier K+ channels., Guo D., J Gen Physiol. April 1, 2003; 121 (4): 261-75.                        

Mechanism underlying bupivacaine inhibition of G protein-gated inwardly rectifying K+ channels., Zhou W., Proc Natl Acad Sci U S A. May 22, 2001; 98 (11): 6482-7.

Tyrosine decaging leads to substantial membrane trafficking during modulation of an inward rectifier potassium channel., Tong Y., J Gen Physiol. February 1, 2001; 117 (2): 103-18.                

Molecular and functional heterogeneity of inward rectifier potassium channels in brain and heart., Kurachi Y., J Card Fail. December 1, 1996; 2 (4 Suppl): S59-62.

Complexity of the regulation of Kir2.1 K+ channels., Ruppersberg JP., Neuropharmacology. January 1, 1996; 35 (7): 887-93.

Susceptibility of cloned K+ channels to reactive oxygen species., Duprat F., Proc Natl Acad Sci U S A. December 5, 1995; 92 (25): 11796-800.

Cloning provides evidence for a family of inward rectifier and G-protein coupled K+ channels in the brain., Lesage F., FEBS Lett. October 10, 1994; 353 (1): 37-42.

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