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Summary Anatomy Item Literature (471) Expression Attributions Wiki
XB-ANAT-3647

Papers associated with neuromere (and hoxa1)

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Genetically programmed retinoic acid deficiency during gastrulation phenocopies most known developmental defects due to acute prenatal alcohol exposure in FASD., Petrelli B., Front Cell Dev Biol. January 1, 2023; 11 1208279.                    

Hox and Pbx factors control retinoic acid synthesis during hindbrain segmentation., Vitobello A., Dev Cell. April 19, 2011; 20 (4): 469-82.  

Retinoid signalling is required for information transfer from mesoderm to neuroectoderm during gastrulation., Lloret-Vilaspasa F., Int J Dev Biol. January 1, 2010; 54 (4): 599-608.                

Knockdown of the complete Hox paralogous group 1 leads to dramatic hindbrain and neural crest defects., McNulty CL., Development. June 1, 2005; 132 (12): 2861-71.                    

Global analysis of RAR-responsive genes in the Xenopus neurula using cDNA microarrays., Arima K., Dev Dyn. February 1, 2005; 232 (2): 414-31.                          

The Meis3 protein and retinoid signaling interact to pattern the Xenopus hindbrain., Dibner C., Dev Biol. July 1, 2004; 271 (1): 75-86.              

Xenopus hindbrain patterning requires retinoid signaling., Kolm PJ., Dev Biol. December 1, 1997; 192 (1): 1-16.              

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