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Summary Anatomy Item Literature (1721) Expression Attributions Wiki
XB-ANAT-1495

Papers associated with portion of organism substance (and pomc)

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Impaired negative feedback and death following acute stress in glucocorticoid receptor knockout Xenopus tropicalis tadpoles., Paul B., Gen Comp Endocrinol. September 15, 2022; 326 114072.      

Melanocortin Receptor 4 Signaling Regulates Vertebrate Limb Regeneration., Zhang M., Dev Cell. August 20, 2018; 46 (4): 397-409.e5.                              

Angiogenesis in the intermediate lobe of the pituitary gland alters its structure and function., Tanaka S., Gen Comp Endocrinol. May 1, 2013; 185 10-8.        

The origins and evolution of vertebrate metamorphosis., Laudet V., Curr Biol. September 27, 2011; 21 (18): R726-37.            

Brain distribution and evidence for both central and neurohormonal actions of cocaine- and amphetamine-regulated transcript peptide in Xenopus laevis., Roubos EW., J Comp Neurol. April 1, 2008; 507 (4): 1622-38.                  

Widespread tissue distribution and diverse functions of corticotropin-releasing factor and related peptides., Boorse GC., Gen Comp Endocrinol. March 1, 2006; 146 (1): 9-18.      

Evidence that urocortin I acts as a neurohormone to stimulate alpha MSH release in the toad Xenopus laevis., Calle M., Dev Biol. April 8, 2005; 1040 (1-2): 14-28.              

Opioid peptides, CRF, and urocortin in cerebrospinal fluid-contacting neurons in Xenopus laevis., Calle M., Ann N Y Acad Sci. April 1, 2005; 1040 249-52.

Timing of metamorphosis and the onset of the negative feedback loop between the thyroid gland and the pituitary is controlled by type II iodothyronine deiodinase in Xenopus laevis., Huang H., Proc Natl Acad Sci U S A. June 19, 2001; 98 (13): 7348-53.          

Corticotropin-releasing factor (CRF)-like immunoreactivity in the gastro-entero-pancreatic endocrine system., Petrusz P., Peptides. January 1, 1984; 5 Suppl 1 71-8.

Evidence that glycosylation of pro-opiocortin and ACTH influences their proteolysis by trypsin and blood proteases., Loh YP., Mol Cell Endocrinol. October 1, 1980; 20 (1): 35-44.

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