Papers associated with portion of organism substance (and kcnj11)

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	A loss-of-function mutation in KCNJ11 causing sulfonylurea-sensitive diabetes in early adult life., Vedovato N., Diabetologia. May 1, 2024; 67 (5): 940-951.
	Beneficial actions of the [A14K] analog of the frog skin peptide PGLa-AM1 in mice with obesity and degenerative diabetes: A mechanistic study., Musale V., Peptides. February 1, 2021; 136 170472.
	Permanent neonatal diabetes: combining sulfonylureas with insulin may be an effective treatment., Misra S., Diabet Med. June 13, 2018;
	Distinct action of the α-glucosidase inhibitor miglitol on SGLT3, enteroendocrine cells, and GLP1 secretion., Lee EY., J Endocrinol. March 1, 2015; 224 (3): 205-14.
	Disease progression and search for monogenic diabetes among children with new onset type 1 diabetes negative for ICA, GAD- and IA-2 Antibodies., Pörksen S., BMC Endocr Disord. September 23, 2010; 10 16.
	Targeting hypertension with a new adenosine triphosphate-sensitive potassium channel opener iptakalim., Pan Z., J Cardiovasc Pharmacol. September 1, 2010; 56 (3): 215-28.
	Open state destabilization by ATP occupancy is mechanism speeding burst exit underlying KATP channel inhibition by ATP., Li L., J Gen Physiol. January 1, 2002; 119 (1): 105-16.
	Truncation of Kir6.2 produces ATP-sensitive K+ channels in the absence of the sulphonylurea receptor., Tucker SJ., Nature. May 8, 1997; 387 (6629): 179-83.

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