Papers associated with portion of organism substance (and kcnj1)

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	Mutation affecting the conserved acidic WNK1 motif causes inherited hyperkalemic hyperchloremic acidosis., Louis-Dit-Picard H., J Clin Invest. December 1, 2020; 130 (12): 6379-6394.
	Development of a selective small-molecule inhibitor of Kir1.1, the renal outer medullary potassium channel., Bhave G., Mol Pharmacol. January 1, 2011; 79 (1): 42-50.
	Hypertension resistance polymorphisms in ROMK (Kir1.1) alter channel function by different mechanisms., Fang L., Am J Physiol Renal Physiol. December 1, 2010; 299 (6): F1359-64.
	Organization of the pronephric kidney revealed by large-scale gene expression mapping., Raciti D., Genome Biol. January 1, 2008; 9 (5): R84.
	[WNK1 and WNK4, new players in salt and water homeostasis], Hadchouel J., Med Sci (Paris). January 1, 2005; 21 (1): 55-60.
	WNK4 regulates apical and basolateral Cl- flux in extrarenal epithelia., Kahle KT., Proc Natl Acad Sci U S A. February 17, 2004; 101 (7): 2064-9.
	WNK4 regulates the balance between renal NaCl reabsorption and K+ secretion., Kahle KT., Nat Genet. December 1, 2003; 35 (4): 372-6.
	Molecular cloning and functional expression of a novel brain-specific inward rectifier potassium channel., Morishige K., FEBS Lett. June 13, 1994; 346 (2-3): 251-6.
	Molecular cloning, functional expression and localization of a novel inward rectifier potassium channel in the rat brain., Koyama H., FEBS Lett. March 21, 1994; 341 (2-3): 303-7.

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