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Summary Anatomy Item Literature (1721) Expression Attributions Wiki
XB-ANAT-1495

Papers associated with portion of organism substance (and abcc8)

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A loss-of-function mutation in KCNJ11 causing sulfonylurea-sensitive diabetes in early adult life., Vedovato N., Diabetologia. May 1, 2024; 67 (5): 940-951.          

A novel high-affinity inhibitor against the human ATP-sensitive Kir6.2 channel., Ramu Y., J Gen Physiol. July 2, 2018; 150 (7): 969-976.              

Disease progression and search for monogenic diabetes among children with new onset type 1 diabetes negative for ICA, GAD- and IA-2 Antibodies., Pörksen S., BMC Endocr Disord. September 23, 2010; 10 16.      

Targeting hypertension with a new adenosine triphosphate-sensitive potassium channel opener iptakalim., Pan Z., J Cardiovasc Pharmacol. September 1, 2010; 56 (3): 215-28.

Expression of complement components coincides with early patterning and organogenesis in Xenopus laevis., McLin VA., Int J Dev Biol. January 1, 2008; 52 (8): 1123-33.                                              

Open state destabilization by ATP occupancy is mechanism speeding burst exit underlying KATP channel inhibition by ATP., Li L., J Gen Physiol. January 1, 2002; 119 (1): 105-16.                    

Truncation of Kir6.2 produces ATP-sensitive K+ channels in the absence of the sulphonylurea receptor., Tucker SJ., Nature. May 8, 1997; 387 (6629): 179-83.

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