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Summary Anatomy Item Literature (3729) Expression Attributions Wiki
XB-ANAT-99

Papers associated with cardiovascular system (and nav1)

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Differential regulation of cardiac sodium channels by intracellular fibroblast growth factors., Angsutararux P., J Gen Physiol. May 1, 2023; 155 (5):


Molecular charge associated with antiarrhythmic actions in a series of amino-2-cyclohexyl ester derivatives., Pugsley MK., Eur J Pharmacol. February 5, 2019; 844 241-252.


Pharmacology of the Nav1.1 domain IV voltage sensor reveals coupling between inactivation gating processes., Osteen JD., Proc Natl Acad Sci U S A. June 27, 2017; 114 (26): 6836-6841.


Splicing misregulation of SCN5A contributes to cardiac-conduction delay and heart arrhythmia in myotonic dystrophy., Freyermuth F., Nat Commun. April 11, 2016; 7 11067.              


Gain-of-function mutation in TASK-4 channels and severe cardiac conduction disorder., Friedrich C., EMBO Mol Med. July 1, 2014; 6 (7): 937-51.              


A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype., Gosselin-Badaroudine P., PLoS One. January 1, 2012; 7 (5): e38331.          


Pharmacological modulation of brain Nav1.2 and cardiac Nav1.5 subtypes by the local anesthetic ropivacaine., Cheng HW., Neurosci Bull. August 1, 2010; 26 (4): 289-96.


Blocking effect of methylflavonolamine on human Na(V)1.5 channels expressed in Xenopus laevis oocytes and on sodium currents in rabbit ventricular myocytes., Fan XR., Acta Pharmacol Sin. March 1, 2010; 31 (3): 297-306.                  


Correlations between clinical and physiological consequences of the novel mutation R878C in a highly conserved pore residue in the cardiac Na+ channel., Zhang Y., Acta Physiol (Oxf). December 1, 2008; 194 (4): 311-23.            


Nav channel mechanosensitivity: activation and inactivation accelerate reversibly with stretch., Morris CE., Biophys J. August 1, 2007; 93 (3): 822-33.


H+ pump-dependent changes in membrane voltage are an early mechanism necessary and sufficient to induce Xenopus tail regeneration., Adams DS., Development. April 1, 2007; 134 (7): 1323-35.          


GLUT8 is dispensable for embryonic development but influences hippocampal neurogenesis and heart function., Membrez M., Mol Cell Biol. June 1, 2006; 26 (11): 4268-76.


Distribution and functional characterization of human Nav1.3 splice variants., Thimmapaya R., Eur J Neurosci. July 1, 2005; 22 (1): 1-9.


Occurrence of a tetrodotoxin-sensitive calcium current in rat ventricular myocytes after long-term myocardial infarction., Alvarez JL., Cardiovasc Res. September 1, 2004; 63 (4): 653-61.

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