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Summary Anatomy Item Literature (3729) Expression Attributions Wiki
XB-ANAT-99

Papers associated with cardiovascular system (and kcnj6)

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A Collision Coupling Model Governs the Activation of Neuronal GIRK1/2 Channels by Muscarinic-2 Receptors., Berlin S., Front Pharmacol. April 7, 2020; 11 1216.                              


Functional diversity of voltage-sensing phosphatases in two urodele amphibians., Mutua J., Physiol Rep. July 16, 2014; 2 (7):                 


Crystal structure of the mammalian GIRK2 K+ channel and gating regulation by G proteins, PIP2, and sodium., Whorton MR., Cell. September 30, 2011; 147 (1): 199-208.


Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants., Kobayashi T., PLoS One. January 1, 2011; 6 (12): e28208.            


Inhibition of G-protein-activated inwardly rectifying K+ channels by the selective norepinephrine reuptake inhibitors atomoxetine and reboxetine., Kobayashi T., Neuropsychopharmacology. June 1, 2010; 35 (7): 1560-9.


Pregnenolone sulfate potentiates the inwardly rectifying K channel Kir2.3., Kobayashi T., PLoS One. July 21, 2009; 4 (7): e6311.              


Inhibition by cocaine of G protein-activated inwardly rectifying K+ channels expressed in Xenopus oocytes., Kobayashi T., Toxicol In Vitro. June 1, 2007; 21 (4): 656-64.


Inhibition of G protein-activated inwardly rectifying K+ channels by the antidepressant paroxetine., Kobayashi T., J Pharmacol Sci. November 1, 2006; 102 (3): 278-87.


Inhibition of G protein-activated inwardly rectifying K+ channels by ifenprodil., Kobayashi T., Neuropsychopharmacology. March 1, 2006; 31 (3): 516-24.


Modulators of G protein-activated inwardly rectifying K+ channels: potentially therapeutic agents for addictive drug users., Kobayashi T., Ann N Y Acad Sci. October 1, 2004; 1025 590-4.


Inhibition of G protein-activated inwardly rectifying K+ channels by various antidepressant drugs., Kobayashi T., Neuropsychopharmacology. October 1, 2004; 29 (10): 1841-51.


Evolving potassium channels by means of yeast selection reveals structural elements important for selectivity., Bichet D., Proc Natl Acad Sci U S A. March 30, 2004; 101 (13): 4441-6.


Inhibition of G protein-activated inwardly rectifying K+ channels by fluoxetine (Prozac)., Kobayashi T., Br J Pharmacol. March 1, 2003; 138 (6): 1119-28.


G-protein mediated gating of inward-rectifier K+ channels., Mark MD., Eur J Biochem. October 1, 2000; 267 (19): 5830-6.


Impermeability of the GIRK2 weaver channel to divalent cations., Hou P., Am J Physiol Cell Physiol. May 1, 2000; 278 (5): C1038-46.


Ethanol opens G-protein-activated inwardly rectifying K+ channels., Kobayashi T., Nat Neurosci. December 1, 1999; 2 (12): 1091-7.


Positive and negative coupling of the metabotropic glutamate receptors to a G protein-activated K+ channel, GIRK, in Xenopus oocytes., Sharon D., J Gen Physiol. April 1, 1997; 109 (4): 477-90.        


Control of channel activity through a unique amino acid residue of a G protein-gated inwardly rectifying K+ channel subunit., Chan KW., Proc Natl Acad Sci U S A. November 26, 1996; 93 (24): 14193-8.


Evidence that neuronal G-protein-gated inwardly rectifying K+ channels are activated by G beta gamma subunits and function as heteromultimers., Kofuji P., Proc Natl Acad Sci U S A. July 3, 1995; 92 (14): 6542-6.

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