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Aberrant connexin26 hemichannels underlying keratitis-ichthyosis-deafness syndrome are potently inhibited by mefloquine. , Levit NA., J Invest Dermatol. April 1, 2015; 135 (4): 1033-1042.
The human Cx26-D50A and Cx26-A88V mutations causing keratitis-ichthyosis-deafness syndrome display increased hemichannel activity. , Mhaske PV., Am J Physiol Cell Physiol. June 15, 2013; 304 (12): C1150-8.
Expression of odorant receptor family, type 2 OR in the aquatic olfactory cavity of amphibian frog Xenopus tropicalis. , Amano T ., PLoS One. January 1, 2012; 7 (4): e33922.
Differentially altered Ca2+ regulation and Ca2+ permeability in Cx26 hemichannels formed by the A40V and G45E mutations that cause keratitis ichthyosis deafness syndrome. , Sánchez HA., J Gen Physiol. July 1, 2010; 136 (1): 47-62.
Connexin mutations causing skin disease and deafness increase hemichannel activity and cell death when expressed in Xenopus oocytes. , Lee JR , Lee JR ., J Invest Dermatol. April 1, 2009; 129 (4): 870-8.
Essential role of CREB family proteins during Xenopus embryogenesis. , Lutz B., Mech Dev. October 1, 1999; 88 (1): 55-66.
A mesoderm-inducing factor is produced by Xenopus cell line. , Smith JC ., Development. January 1, 1987; 99 (1): 3-14.