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Modeling endoderm development and disease in Xenopus. , Edwards NA ., Curr Top Dev Biol. January 1, 2021; 145 61-90.
Bioelectric signalling via potassium channels: a mechanism for craniofacial dysmorphogenesis in KCNJ2-associated Andersen-Tawil Syndrome. , Adams DS ., J Physiol. June 15, 2016; 594 (12): 3245-70.
Xenopus as a model system for studying pancreatic development and diabetes. , Kofent J., Semin Cell Dev Biol. March 1, 2016; 51 106-16.
Recessive mutations in PCBD1 cause a new type of early-onset diabetes. , Simaite D., Diabetes. October 1, 2014; 63 (10): 3557-64.
A gating mutation at the internal mouth of the Kir6.2 pore is associated with DEND syndrome. , Proks P., EMBO Rep. May 1, 2005; 6 (5): 470-5.
K(ATP) channel activity is required for hatching in Xenopus embryos. , Cheng SM., Dev Dyn. December 1, 2002; 225 (4): 588-91.
Mutations within the P-loop of Kir6.2 modulate the intraburst kinetics of the ATP-sensitive potassium channel. , Proks P., J Gen Physiol. October 1, 2001; 118 (4): 341-53.