Papers associated with vestibule

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	Molecular rearrangements of the extracellular vestibule in NMDAR channels during gating., Sobolevsky AI., Neuron. January 3, 2002; 33 (1): 75-85.
	Designed peptide analogues of the potassium channel blocker ShK toxin., Lanigan MD., Biochemistry. December 25, 2001; 40 (51): 15528-37.
	Mutation K448E in the external loop 5 of rat GABA transporter rGAT1 induces pH sensitivity and alters substrate interactions., Forlani G., J Physiol. October 15, 2001; 536 (Pt 2): 479-94.
	Exploration of the pore structure of a peptide-gated Na+ channel., Poët M., EMBO J. October 15, 2001; 20 (20): 5595-602.
	CFTR: covalent and noncovalent modification suggests a role for fixed charges in anion conduction., Smith SS., J Gen Physiol. October 1, 2001; 118 (4): 407-31.
	Antagonism of nicotinic acetylcholine receptors by inhibitors of monoamine uptake., López-Valdés HE., Mol Psychiatry. September 1, 2001; 6 (5): 511-9.
	The selectivity filter of the voltage-gated sodium channel is involved in channel activation., Hilber K., J Biol Chem. July 27, 2001; 276 (30): 27831-9.
	Channel-lining residues of the AMPA receptor M2 segment: structural environment of the Q/R site and identification of the selectivity filter., Kuner T., J Neurosci. June 15, 2001; 21 (12): 4162-72.
	Conformational changes in S6 coupled to the opening of cyclic nucleotide-gated channels., Flynn GE., Neuron. June 1, 2001; 30 (3): 689-98.
	Functional properties of a truncated recombinant GIRK5 potassium channel., Salvador C., Biochim Biophys Acta. May 2, 2001; 1512 (1): 135-47.
	Structural and gating changes of the sodium channel induced by mutation of a residue in the upper third of IVS6, creating an external access path for local anesthetics., Sunami A., Mol Pharmacol. April 1, 2001; 59 (4): 684-91.
	Titration of tertiapin-Q inhibition of ROMK1 channels by extracellular protons., Ramu Y., Biochemistry. March 27, 2001; 40 (12): 3601-5.
	Control of rectification and permeation by two distinct sites after the second transmembrane region in Kir2.1 K+ channel., Kubo Y., J Physiol. March 15, 2001; 531 (Pt 3): 645-60.
	Specific neosaxitoxin interactions with the Na+ channel outer vestibule determined by mutant cycle analysis., Penzotti JL., Biophys J. February 1, 2001; 80 (2): 698-706.
	Modulation of alpha2beta4 neuronal nicotinic acetylcholine receptors by zinc., García-Colunga J., Neuroreport. January 22, 2001; 12 (1): 147-50.
	Gating induces a conformational change in the outer vestibule of ENaC., Snyder PM., J Gen Physiol. December 1, 2000; 116 (6): 781-90.
	The role of positively charged amino acids in ATP recognition by human P2X(1) receptors., Ennion S., J Biol Chem. September 22, 2000; 275 (38): 29361-7.
	Structure of the amino terminus of a gap junction protein., Purnick PE., Arch Biochem Biophys. September 15, 2000; 381 (2): 181-90.
	Glycoside binding and translocation in Na(+)-dependent glucose cotransporters: comparison of SGLT1 and SGLT3., Díez-Sampedro A., J Membr Biol. July 15, 2000; 176 (2): 111-7.
	Trapping of a methanesulfonanilide by closure of the HERG potassium channel activation gate., Mitcheson JS., J Gen Physiol. March 1, 2000; 115 (3): 229-40.
	A critical residue for isoform difference in tetrodotoxin affinity is a molecular determinant of the external access path for local anesthetics in the cardiac sodium channel., Sunami A., Proc Natl Acad Sci U S A. February 29, 2000; 97 (5): 2326-31.
	Deletion of the S3-S4 linker in the Shaker potassium channel reveals two quenching groups near the outside of S4., Sørensen JB., J Gen Physiol. February 1, 2000; 115 (2): 209-22.
	Gating and flickery block differentially affected by rubidium in homomeric KCNQ1 and heteromeric KCNQ1/KCNE1 potassium channels., Pusch M., Biophys J. January 1, 2000; 78 (1): 211-26.
	Identification of transduction elements for benzodiazepine modulation of the GABA(A) receptor: three residues are required for allosteric coupling., Boileau AJ., J Neurosci. December 1, 1999; 19 (23): 10213-20.
	Inward rectifier potassium channel Kir 2.3 is inhibited by internal sulfhydryl modification., Radeke CM., Neuroreport. November 8, 1999; 10 (16): 3277-82.
	Mechanisms of inward-rectifier K+ channel inhibition by tertiapin-Q., Jin W., Biochemistry. October 26, 1999; 38 (43): 14294-301.
	Cytoplasmic amino and carboxyl domains form a wide intracellular vestibule in an inwardly rectifying potassium channel., Lu T., Proc Natl Acad Sci U S A. August 17, 1999; 96 (17): 9926-31.
	Bicuculline block of small-conductance calcium-activated potassium channels., Khawaled R., Pflugers Arch. August 1, 1999; 438 (3): 314-21.
	A marine snail neurotoxin shares with scorpion toxins a convergent mechanism of blockade on the pore of voltage-gated K channels., García E., J Gen Physiol. July 1, 1999; 114 (1): 141-57.
	On the molecular basis of ion permeation in the epithelial Na+ channel., Kellenberger S., J Gen Physiol. July 1, 1999; 114 (1): 13-30.
	Tonic and phasic tetrodotoxin block of sodium channels with point mutations in the outer pore region., Boccaccio A., Biophys J. July 1, 1999; 77 (1): 229-40.
	Inactivation gating of Kv4 potassium channels: molecular interactions involving the inner vestibule of the pore., Jerng HH., J Gen Physiol. May 1, 1999; 113 (5): 641-60.
	NMDAR channel segments forming the extracellular vestibule inferred from the accessibility of substituted cysteines., Beck C., Neuron. March 1, 1999; 22 (3): 559-70.
	Ultra-slow inactivation in mu1 Na+ channels is produced by a structural rearrangement of the outer vestibule., Todt H., Biophys J. March 1, 1999; 76 (3): 1335-45.
	Mutation of structural determinants lining the N-methyl-D-aspartate receptor channel differentially affects phencyclidine block and spermine potentiation and block., Zheng X., Neuroscience. January 1, 1999; 93 (1): 125-34.
	Differences in saxitoxin and tetrodotoxin binding revealed by mutagenesis of the Na+ channel outer vestibule., Penzotti JL., Biophys J. December 1, 1998; 75 (6): 2647-57.
	Block of the Kir2.1 channel pore by alkylamine analogues of endogenous polyamines., Pearson WL., J Gen Physiol. September 1, 1998; 112 (3): 351-63.
	Predominant interactions between mu-conotoxin Arg-13 and the skeletal muscle Na+ channel localized by mutant cycle analysis., Chang NS., Biochemistry. March 31, 1998; 37 (13): 4407-19.
	A novel toxin form the scorpion Androctonus australis blocks Shaker K+ channels expressed in Xenopus oocytes., Pisciotta M., Biochem Biophys Res Commun. January 14, 1998; 242 (2): 287-91.
	[Barium ion blockade on an inward rectifying potassium current in oocytes of the frog Xenopus laevis]., Gamboa R., Arch Inst Cardiol Mex. January 1, 1998; 68 (3): 206-13.
	Solution structure and proposed binding mechanism of a novel potassium channel toxin kappa-conotoxin PVIIA., Scanlon MJ., Structure. December 15, 1997; 5 (12): 1585-97.
	Identification of an amino acid residue that lies between the exofacial vestibule and exofacial substrate-binding site of the Glut1 sugar permeation pathway., Mueckler M., J Biol Chem. November 28, 1997; 272 (48): 30141-6.
	The N-terminal domain of a K+ channel beta subunit increases the rate of C-type inactivation from the cytoplasmic side of the channel., Morales MJ., Proc Natl Acad Sci U S A. December 24, 1996; 93 (26): 15119-23.
	The signature sequence of voltage-gated potassium channels projects into the external vestibule., Aiyar J., J Biol Chem. December 6, 1996; 271 (49): 31013-6.
	The initiation of the muscle action potential., Neumann E., Arch Physiol Biochem. October 1, 1996; 104 (6): 731-44.
	Probing the pore region of recombinant N-methyl-D-aspartate channels using external and internal magnesium block., Kupper J., Proc Natl Acad Sci U S A. August 6, 1996; 93 (16): 8648-53.
	Depth asymmetries of the pore-lining segments of the Na+ channel revealed by cysteine mutagenesis., Chiamvimonvat N., Neuron. May 1, 1996; 16 (5): 1037-47.
	Permeation properties and differential expression across the auditory receptor epithelium of an inward rectifier K+ channel cloned from the chick inner ear., Navaratnam DS., J Biol Chem. August 18, 1995; 270 (33): 19238-45.
	Specificity for block by saxitoxin and divalent cations at a residue which determines sensitivity of sodium channel subtypes to guanidinium toxins., Favre I., J Gen Physiol. August 1, 1995; 106 (2): 203-29.
	Side-chain accessibilities in the pore of a K+ channel probed by sulfhydryl-specific reagents after cysteine-scanning mutagenesis., Kürz LL., Biophys J. March 1, 1995; 68 (3): 900-5.

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