Papers associated with heart (and ikzf1)

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	Verapamil inhibits Kir2.3 channels by binding to the pore and interfering with PIP2 binding., Xynogalos P., Naunyn Schmiedebergs Arch Pharmacol. April 1, 2023; 396 (4): 659-667.
	Facilitation of IKr current by some hERG channel blockers suppresses early afterdepolarizations., Furutani K., J Gen Physiol. February 4, 2019; 151 (2): 214-230.
	Inhibition of inwardly rectifying Kir2.x channels by the novel anti-cancer agent gambogic acid depends on both pore block and PIP2 interference., Scherer D., Naunyn Schmiedebergs Arch Pharmacol. July 1, 2017; 390 (7): 701-710.
	Dual Mechanism for Inhibition of Inwardly Rectifying Kir2.x Channels by Quinidine Involving Direct Pore Block and PIP2-interference., Koepple C., J Pharmacol Exp Ther. May 1, 2017; 361 (2): 209-218.
	Role of plasma membrane-associated AKAPs for the regulation of cardiac IK1 current by protein kinase A., Seyler C., Naunyn Schmiedebergs Arch Pharmacol. May 1, 2017; 390 (5): 493-503.
	Inhibition of Cardiac Kir Current (IK1) by Protein Kinase C Critically Depends on PKCβ and Kir2.2., Scherer D., PLoS One. May 23, 2016; 11 (5): e0156181.
	Class III antiarrhythmic drug dronedarone inhibits cardiac inwardly rectifying Kir2.1 channels through binding at residue E224., Xynogalos P., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2014; 387 (12): 1153-61.
	Puerarin: a novel antagonist to inward rectifier potassium channel (IK1)., Zhang H., Mol Cell Biochem. June 1, 2011; 352 (1-2): 117-23.
	Integrative genomic analyses on Ikaros and its expression related to solid cancer prognosis., Yang L., Oncol Rep. August 1, 2010; 24 (2): 571-7.
	Regulation of cardiac inwardly rectifying potassium current IK1 and Kir2.x channels by endothelin-1., Kiesecker C., J Mol Med (Berl). January 1, 2006; 84 (1): 46-56.
	Human cardiac inwardly rectifying current IKir2.2 is upregulated by activation of protein kinase A., Zitron E., Cardiovasc Res. August 15, 2004; 63 (3): 520-7.
	Human cardiac inwardly-rectifying K+ channel Kir(2.1b) is inhibited by direct protein kinase C-dependent regulation in human isolated cardiomyocytes and in an expression system., Karle CA., Circulation. September 17, 2002; 106 (12): 1493-9.
	Role of the thrombopoietin (TPO)/Mpl system: c-Mpl-like molecule/TPO signaling enhances early hematopoiesis in Xenopus laevis., Kakeda M., Dev Growth Differ. February 1, 2002; 44 (1): 63-75.
	Inhibitory effects of berberine on IK1, IK, and HERG channels of cardiac myocytes., Li BX., Acta Pharmacol Sin. February 1, 2001; 22 (2): 125-31.
	Inhibition of IKs channels by HMR 1556., Gögelein H., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2000; 362 (6): 480-8.
	Unitary current through the inward rectifier K+ channel cloned from rabbit heart--comparison with the native K+ channel., Nagashima M., J Mol Cell Cardiol. May 1, 1996; 28 (5): 957-65.
	Cloning and functional expression of a human gene, hIRK1, encoding the heart inward rectifier K+-channel., Wood LS., Gene. October 3, 1995; 163 (2): 313-7.
	Cloning and functional expression of an inwardly rectifying K+ channel from human atrium., Wible BA., Circ Res. March 1, 1995; 76 (3): 343-50.
	The novel class III antiarrhythmics NE-10064 and NE-10133 inhibit IsK channels expressed in Xenopus oocytes and IKs in guinea pig cardiac myocytes., Busch AE., Biochem Biophys Res Commun. July 15, 1994; 202 (1): 265-70.

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