Papers associated with gonad (and kcne1)

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	Pharmacological rescue of specific long QT variants of KCNQ1/KCNE1 channels., Zou X., Front Physiol. January 1, 2022; 13 902224.
	KCNQ1 rescues TMC1 plasma membrane expression but not mechanosensitive channel activity., Harkcom WT., J Cell Physiol. August 1, 2019; 234 (8): 13361-13369.
	Strong activation of ether-à-go-go-related gene 1 K+ channel isoforms by NS1643 in human embryonic kidney 293 and Chinese hamster ovary cells., Schuster AM., Mol Pharmacol. November 1, 2011; 80 (5): 930-42.
	Sudden infant death syndrome and long QT syndrome: an epidemiological and genetic study., Wedekind H., Int J Legal Med. May 1, 2006; 120 (3): 129-37.
	KCNE2 modulates current amplitudes and activation kinetics of HCN4: influence of KCNE family members on HCN4 currents., Decher N., Pflugers Arch. September 1, 2003; 446 (6): 633-40.
	Modulation of homomeric and heteromeric KCNQ1 channels by external acidification., Peretz A., J Physiol. December 15, 2002; 545 (3): 751-66.
	A comparison of currents carried by HERG, with and without coexpression of MiRP1, and the native rapid delayed rectifier current. Is MiRP1 the missing link?, Weerapura M., J Physiol. April 1, 2002; 540 (Pt 1): 15-27.
	KCNE1 reverses the response of the human K+ channel KCNQ1 to cytosolic pH changes and alters its pharmacology and sensitivity to temperature., Unsöld B., Pflugers Arch. December 1, 2000; 441 (2-3): 368-78.
	Molecular and functional characterization of s-KCNQ1 potassium channel from rectal gland of Squalus acanthias., Waldegger S., Pflugers Arch. January 1, 1999; 437 (2): 298-304.
	Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.

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