Papers associated with tail vasculature (and cav2)

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	Differential zinc permeation and blockade of L-type Ca2+ channel isoforms Cav1.2 and Cav1.3., Park SJ., Biochim Biophys Acta. October 1, 2015; 1848 (10 Pt A): 2092-100.
	Regulation of neuronal high-voltage activated Ca(V)2 Ca(2+) channels by the small GTPase RhoA., Rousset M., Neuropharmacology. October 1, 2015; 97 201-9.
	Lipid modulation of calcium flux through CaV2.3 regulates acrosome exocytosis and fertilization., Cohen R., Dev Cell. February 10, 2014; 28 (3): 310-21.
	N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2., Page KM., J Biol Chem. January 8, 2010; 285 (2): 835-44.
	RGK GTPase-dependent CaV2.1 Ca2+ channel inhibition is independent of CaVbeta-subunit-induced current potentiation., Leyris JP., FASEB J. August 1, 2009; 23 (8): 2627-38.
	Use-dependent block of voltage-gated Cav2.1 Ca2+ channels by petasins and eudesmol isomers., Horak S., J Pharmacol Exp Ther. July 1, 2009; 330 (1): 220-6.
	Disruption of the IS6-AID linker affects voltage-gated calcium channel inactivation and facilitation., Findeisen F., J Gen Physiol. March 1, 2009; 133 (3): 327-43.
	Neurite outgrowth and in vivo sensory innervation mediated by a Ca(V)2.2-laminin beta 2 stop signal., Sann SB., J Neurosci. March 5, 2008; 28 (10): 2366-74.
	Dominant-negative effects of human P/Q-type Ca2+ channel mutations associated with episodic ataxia type 2., Jeng CJ., Am J Physiol Cell Physiol. April 1, 2006; 290 (4): C1209-20.
	Functional architecture of the inner pore of a voltage-gated Ca2+ channel., Zhen XG., J Gen Physiol. September 1, 2005; 126 (3): 193-204.
	A Purkinje cell specific GoLoco domain protein, L7/Pcp-2, modulates receptor-mediated inhibition of Cav2.1 Ca2+ channels in a dose-dependent manner., Kinoshita-Kawada M., Brain Res Mol Brain Res. December 6, 2004; 132 (1): 73-86.
	Ca2+-dependent interaction of BAPTA with phospholipids., Rousset M., FEBS Lett. October 8, 2004; 576 (1-2): 41-5.
	Overexpressed Ca(v)beta3 inhibits N-type (Cav2.2) calcium channel currents through a hyperpolarizing shift of ultra-slow and closed-state inactivation., Yasuda T., J Gen Physiol. April 1, 2004; 123 (4): 401-16.
	The interaction between the I-II loop and the III-IV loop of Cav2.1 contributes to voltage-dependent inactivation in a beta -dependent manner., Geib S., J Biol Chem. March 22, 2002; 277 (12): 10003-13.
	Molecular determinants of inactivation within the I-II linker of alpha1E (CaV2.3) calcium channels., Berrou L., Biophys J. January 1, 2001; 80 (1): 215-28.

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