Papers associated with tail vasculature (and cacna1b)

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	Differential zinc permeation and blockade of L-type Ca2+ channel isoforms Cav1.2 and Cav1.3., Park SJ., Biochim Biophys Acta. October 1, 2015; 1848 (10 Pt A): 2092-100.
	Endogenous gradients of resting potential instructively pattern embryonic neural tissue via Notch signaling and regulation of proliferation., Pai VP., J Neurosci. March 11, 2015; 35 (10): 4366-85.
	Transmembrane voltage potential is an essential cellular parameter for the detection and control of tumor development in a Xenopus model., Chernet BT., Dis Model Mech. May 1, 2013; 6 (3): 595-607.
	N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2., Page KM., J Biol Chem. January 8, 2010; 285 (2): 835-44.
	Neurite outgrowth and in vivo sensory innervation mediated by a Ca(V)2.2-laminin beta 2 stop signal., Sann SB., J Neurosci. March 5, 2008; 28 (10): 2366-74.
	The importance of occupancy rather than affinity of CaV(beta) subunits for the calcium channel I-II linker in relation to calcium channel function., Butcher AJ., J Physiol. July 15, 2006; 574 (Pt 2): 387-98.
	Overexpressed Ca(v)beta3 inhibits N-type (Cav2.2) calcium channel currents through a hyperpolarizing shift of ultra-slow and closed-state inactivation., Yasuda T., J Gen Physiol. April 1, 2004; 123 (4): 401-16.
	The novel product of a five-exon stargazin-related gene abolishes Ca(V)2.2 calcium channel expression., Moss FJ., EMBO J. April 2, 2002; 21 (7): 1514-23.
	Evidence for two concentration-dependent processes for beta-subunit effects on alpha1B calcium channels., Cantí C., Biophys J. September 1, 2001; 81 (3): 1439-51.

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