Papers associated with tail vasculature (and cacna1a)

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	N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2., Page KM., J Biol Chem. January 8, 2010; 285 (2): 835-44.
	RGK GTPase-dependent CaV2.1 Ca2+ channel inhibition is independent of CaVbeta-subunit-induced current potentiation., Leyris JP., FASEB J. August 1, 2009; 23 (8): 2627-38.
	Use-dependent block of voltage-gated Cav2.1 Ca2+ channels by petasins and eudesmol isomers., Horak S., J Pharmacol Exp Ther. July 1, 2009; 330 (1): 220-6.
	Disruption of the IS6-AID linker affects voltage-gated calcium channel inactivation and facilitation., Findeisen F., J Gen Physiol. March 1, 2009; 133 (3): 327-43.
	Impaired plasma membrane targeting or protein stability by certain ATP1A2 mutations identified in sporadic or familial hemiplegic migraine., Tavraz NN., Channels (Austin). January 1, 2009; 3 (2): 82-7.
	Functional architecture of the inner pore of a voltage-gated Ca2+ channel., Zhen XG., J Gen Physiol. September 1, 2005; 126 (3): 193-204.
	A Purkinje cell specific GoLoco domain protein, L7/Pcp-2, modulates receptor-mediated inhibition of Cav2.1 Ca2+ channels in a dose-dependent manner., Kinoshita-Kawada M., Brain Res Mol Brain Res. December 6, 2004; 132 (1): 73-86.
	The interaction between the I-II loop and the III-IV loop of Cav2.1 contributes to voltage-dependent inactivation in a beta -dependent manner., Geib S., J Biol Chem. March 22, 2002; 277 (12): 10003-13.

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