Papers associated with heart (and kcnj3)

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	A revised mechanism of action of hyperaldosteronism-linked mutations in cytosolic domains of GIRK4 (KCNJ5)., Shalomov B., J Physiol. March 1, 2022; 600 (6): 1419-1437.
	A novel ion conducting route besides the central pore in an inherited mutant of G-protein-gated inwardly rectifying K+ channel., Chen IS., J Physiol. February 1, 2022; 600 (3): 603-622.
	A Collision Coupling Model Governs the Activation of Neuronal GIRK1/2 Channels by Muscarinic-2 Receptors., Berlin S., Front Pharmacol. April 7, 2020; 11 1216.
	Non-sedating antihistamines block G-protein-gated inwardly rectifying K+ channels., Chen IS., Br J Pharmacol. September 1, 2019; 176 (17): 3161-3179.
	Mutant KCNJ3 and KCNJ5 Potassium Channels as Novel Molecular Targets in Bradyarrhythmias and Atrial Fibrillation., Yamada N., Circulation. April 30, 2019; 139 (18): 2157-2169.
	Familial Sinus Node Disease Caused by a Gain of GIRK (G-Protein Activated Inwardly Rectifying K+ Channel) Channel Function., Kuß J., Circ Genom Precis Med. January 1, 2019; 12 (1): e002238.
	A Mutation in the G-Protein Gene GNB2 Causes Familial Sinus Node and Atrioventricular Conduction Dysfunction., Stallmeyer B., Circ Res. May 12, 2017; 120 (10): e33-e44.
	The polarization of the G-protein activated potassium channel GIRK5 to the vegetal pole of Xenopus laevis oocytes is driven by a di-leucine motif., Díaz-Bello B., PLoS One. May 15, 2013; 8 (5): e64096.
	Crystal structure of the mammalian GIRK2 K+ channel and gating regulation by G proteins, PIP2, and sodium., Whorton MR., Cell. September 30, 2011; 147 (1): 199-208.
	Four and a half LIM protein 1C (FHL1C): a binding partner for voltage-gated potassium channel K(v1.5)., Poparic I., PLoS One. January 1, 2011; 6 (10): e26524.
	Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants., Kobayashi T., PLoS One. January 1, 2011; 6 (12): e28208.
	Gating of a G protein-sensitive mammalian Kir3.1 prokaryotic Kir channel chimera in planar lipid bilayers., Leal-Pinto E., J Biol Chem. December 17, 2010; 285 (51): 39790-800.
	Inhibition of G-protein-activated inwardly rectifying K+ channels by the selective norepinephrine reuptake inhibitors atomoxetine and reboxetine., Kobayashi T., Neuropsychopharmacology. June 1, 2010; 35 (7): 1560-9.
	Inhibitory effects of the antiepileptic drug ethosuximide on G protein-activated inwardly rectifying K+ channels., Kobayashi T., Neuropharmacology. February 1, 2009; 56 (2): 499-506.
	Characterizations of a loss-of-function mutation in the Kir3.4 channel subunit., Calloe K., Biochem Biophys Res Commun. December 28, 2007; 364 (4): 889-95.
	Inhibition by cocaine of G protein-activated inwardly rectifying K+ channels expressed in Xenopus oocytes., Kobayashi T., Toxicol In Vitro. June 1, 2007; 21 (4): 656-64.
	RGS3 and RGS4 differentially associate with G protein-coupled receptor-Kir3 channel signaling complexes revealing two modes of RGS modulation. Precoupling and collision coupling., Jaén C., J Biol Chem. November 10, 2006; 281 (45): 34549-60.
	Inhibition of G protein-activated inwardly rectifying K+ channels by the antidepressant paroxetine., Kobayashi T., J Pharmacol Sci. November 1, 2006; 102 (3): 278-87.
	Inhibition of G protein-activated inwardly rectifying K+ channels by ifenprodil., Kobayashi T., Neuropsychopharmacology. March 1, 2006; 31 (3): 516-24.
	Mutation of critical GIRK subunit residues disrupts N- and C-termini association and channel function., Sarac R., J Neurosci. February 16, 2005; 25 (7): 1836-46.
	Molecular cloning and characterization of a new RGS protein of Medaka., Itoh M., Gene. January 31, 2005; 345 (2): 165-71.
	Modulators of G protein-activated inwardly rectifying K+ channels: potentially therapeutic agents for addictive drug users., Kobayashi T., Ann N Y Acad Sci. October 1, 2004; 1025 590-4.
	Inhibition of G protein-activated inwardly rectifying K+ channels by various antidepressant drugs., Kobayashi T., Neuropsychopharmacology. October 1, 2004; 29 (10): 1841-51.
	Measuring the modulatory effects of RGS proteins on GIRK channels., Doupnik CA., Methods Enzymol. January 1, 2004; 389 131-54.
	Inhibition of G protein-activated inwardly rectifying K+ channels by fluoxetine (Prozac)., Kobayashi T., Br J Pharmacol. March 1, 2003; 138 (6): 1119-28.
	Cloning and characterization of G protein-gated inward rectifier K+ channel (GIRK1) isoforms from heart and brain., Zhu L., J Mol Neurosci. February 1, 2001; 16 (1): 21-32.
	G-protein mediated gating of inward-rectifier K+ channels., Mark MD., Eur J Biochem. October 1, 2000; 267 (19): 5830-6.
	Heterologous facilitation of G protein-activated K(+) channels by beta-adrenergic stimulation via cAMP-dependent protein kinase., Müllner C., J Gen Physiol. May 1, 2000; 115 (5): 547-58.
	Kir3.1/3.2 encodes an I(KACh)-like current in gastrointestinal myocytes., Bradley KK., Am J Physiol Gastrointest Liver Physiol. February 1, 2000; 278 (2): G289-96.
	Ethanol opens G-protein-activated inwardly rectifying K+ channels., Kobayashi T., Nat Neurosci. December 1, 1999; 2 (12): 1091-7.
	Probing the G-protein regulation of GIRK1 and GIRK4, the two subunits of the KACh channel, using functional homomeric mutants., Vivaudou M., J Biol Chem. December 12, 1997; 272 (50): 31553-60.
	RGS proteins reconstitute the rapid gating kinetics of gbetagamma-activated inwardly rectifying K+ channels., Doupnik CA., Proc Natl Acad Sci U S A. September 16, 1997; 94 (19): 10461-6.
	Cloning and characterization of Kir3.1 (GIRK1) C-terminal alternative splice variants., Nelson CS., Brain Res Mol Brain Res. June 1, 1997; 46 (1-2): 185-96.
	Inwardly rectifying potassium channels: their molecular heterogeneity and function., Isomoto S., Jpn J Physiol. February 1, 1997; 47 (1): 11-39.
	Control of channel activity through a unique amino acid residue of a G protein-gated inwardly rectifying K+ channel subunit., Chan KW., Proc Natl Acad Sci U S A. November 26, 1996; 93 (24): 14193-8.
	Cloning of a G protein-activated inwardly rectifying potassium channel from human cerebellum., Schoots O., Brain Res Mol Brain Res. July 1, 1996; 39 (1-2): 23-30.
	Specificity of coupling of muscarinic receptor isoforms to a novel chick inward-rectifying acetylcholine-sensitive K+ channel., Gadbut AP., J Biol Chem. March 15, 1996; 271 (11): 6398-402.
	Localization and interaction of epitope-tagged GIRK1 and CIR inward rectifier K+ channel subunits., Kennedy ME., Neuropharmacology. January 1, 1996; 35 (7): 831-9.
	Identification of domains conferring G protein regulation on inward rectifier potassium channels., Kunkel MT., Cell. November 3, 1995; 83 (3): 443-9.
	Evidence that direct binding of G beta gamma to the GIRK1 G protein-gated inwardly rectifying K+ channel is important for channel activation., Huang CL., Neuron. November 1, 1995; 15 (5): 1133-43.
	Evidence that neuronal G-protein-gated inwardly rectifying K+ channels are activated by G beta gamma subunits and function as heteromultimers., Kofuji P., Proc Natl Acad Sci U S A. July 3, 1995; 92 (14): 6542-6.
	Cloning and functional characterization of a novel ATP-sensitive potassium channel ubiquitously expressed in rat tissues, including pancreatic islets, pituitary, skeletal muscle, and heart., Inagaki N., J Biol Chem. March 17, 1995; 270 (11): 5691-4.
	The G-protein-gated atrial K+ channel IKACh is a heteromultimer of two inwardly rectifying K(+)-channel proteins., Krapivinsky G., Nature. March 9, 1995; 374 (6518): 135-41.
	Activation of the cloned muscarinic potassium channel by G protein beta gamma subunits., Reuveny E., Nature. July 14, 1994; 370 (6485): 143-6.
	Primary structure and functional expression of a rat G-protein-coupled muscarinic potassium channel., Kubo Y., Nature. August 26, 1993; 364 (6440): 802-6.

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