Click here to close Hello! We notice that you are using Internet Explorer, which is not supported by Xenbase and may cause the site to display incorrectly. We suggest using a current version of Chrome, FireFox, or Safari.

Summary Anatomy Item Literature (2231) Expression Attributions Wiki
XB-ANAT-3282

Papers associated with posterior hypothalamus (and kcnj2)

Limit to papers also referencing gene:
Show all posterior hypothalamus papers
???pagination.result.count???

???pagination.result.page??? 1

Sort Newest To Oldest Sort Oldest To Newest

Genetically induced dysfunctions of Kir2.1 channels: implications for short QT3 syndrome and autism-epilepsy phenotype., Ambrosini E., Hum Mol Genet. September 15, 2014; 23 (18): 4875-86.                      


A splice variant of the two-pore domain potassium channel TREK-1 with only one pore domain reduces the surface expression of full-length TREK-1 channels., Rinné S., Pflugers Arch. August 1, 2014; 466 (8): 1559-70.


Interactions of external K+ and internal blockers in a weak inward-rectifier K+ channel., Yang L., J Gen Physiol. November 1, 2012; 140 (5): 529-40.                


Selection of inhibitor-resistant viral potassium channels identifies a selectivity filter site that affects barium and amantadine block., Chatelain FC., PLoS One. October 16, 2009; 4 (10): e7496.                


KCNE1 and KCNE3 beta-subunits regulate membrane surface expression of Kv12.2 K(+) channels in vitro and form a tripartite complex in vivo., Clancy SM., PLoS One. July 22, 2009; 4 (7): e6330.          


Regulation of Kir channels by intracellular pH and extracellular K(+): mechanisms of coupling., Dahlmann A., J Gen Physiol. April 1, 2004; 123 (4): 441-54.                          


Gastric parietal cell secretory membrane contains PKA- and acid-activated Kir2.1 K+ channels., Malinowska DH., Am J Physiol Cell Physiol. March 1, 2004; 286 (3): C495-506.


Mechanism of rectification in inward-rectifier K+ channels., Guo D., J Gen Physiol. April 1, 2003; 121 (4): 261-75.                        


IRK1 inward rectifier K(+) channels exhibit no intrinsic rectification., Guo D., J Gen Physiol. October 1, 2002; 120 (4): 539-51.                            


Kinetics of inward-rectifier K+ channel block by quaternary alkylammonium ions. dimension and properties of the inner pore., Guo D., J Gen Physiol. May 1, 2001; 117 (5): 395-406.                          


Primary structure, developmental expression and functional properties of an inward rectifier K+ channel of the tunicate., Murata Y., Recept Channels. January 1, 2001; 7 (5): 387-99.


Pore block versus intrinsic gating in the mechanism of inward rectification in strongly rectifying IRK1 channels., Guo D., J Gen Physiol. October 1, 2000; 116 (4): 561-8.              


Molecular cloning and expression of an inwardly rectifying K(+) channel from bovine corneal endothelial cells., Yang D., Invest Ophthalmol Vis Sci. September 1, 2000; 41 (10): 2936-44.


Mechanism of IRK1 channel block by intracellular polyamines., Guo D., J Gen Physiol. June 1, 2000; 115 (6): 799-814.                          


Kir2.1 encodes the inward rectifier potassium channel in rat arterial smooth muscle cells., Bradley KK., J Physiol. March 15, 1999; 515 ( Pt 3) 639-51.


Molecular characterization of an inwardly rectifying K+ channel from HeLa cells., Klein H., J Membr Biol. January 1, 1999; 167 (1): 43-52.


A conserved arginine residue in the pore region of an inward rectifier K channel (IRK1) as an external barrier for cationic blockers., Sabirov RZ., J Gen Physiol. December 1, 1997; 110 (6): 665-77.                  

???pagination.result.page??? 1