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Characterization of Na+ currents regulating intrinsic excitability of optic tectal neurons. , Thompson AC., Life Sci Alliance. January 1, 2024; 7 (1):
Identification of SCN5a p.C335R Variant in a Large Family with Dilated Cardiomyopathy and Conduction Disease. , Sedaghat-Hamedani F., Int J Mol Sci. November 30, 2021; 22 (23):
Functional cross-talk between phosphorylation and disease-causing mutations in the cardiac sodium channel Nav1.5. , Galleano I., Proc Natl Acad Sci U S A. August 17, 2021; 118 (33):
Functional and Structural Characterization of ClC-1 and Nav1.4 Channels Resulting from CLCN1 and SCN4A Mutations Identified Alone and Coexisting in Myotonic Patients. , Brenes O., Cells. February 11, 2021; 10 (2):
Heterologous functional expression of ascidian Nav1 channels and close relationship with the evolutionary ancestor of vertebrate Nav channels. , Kawai T., J Biol Chem. January 1, 2021; 296 100783.
Uncoupling sodium channel dimers restores the phenotype of a pain-linked Nav 1.7 channel mutation. , Rühlmann AH., Br J Pharmacol. October 1, 2020; 177 (19): 4481-4496.
Carvacrol inhibits the neuronal voltage-gated sodium channels Nav1.2, Nav1.6, Nav1.3, Nav1.7, and Nav1.8 expressed in Xenopus oocytes with different potencies. , Horishita T., J Pharmacol Sci. April 1, 2020; 142 (4): 140-147.
Polyunsaturated fatty acid analogues differentially affect cardiac NaV, CaV, and KV channels through unique mechanisms. , Bohannon BM., Elife. March 24, 2020; 9
Gating control of the cardiac sodium channel Nav1.5 by its β3-subunit involves distinct roles for a transmembrane glutamic acid and the extracellular domain. , Salvage SC., J Biol Chem. December 20, 2019; 294 (51): 19752-19763.
The mechanosensitive ion channel TRAAK is localized to the mammalian node of Ranvier. , Brohawn SG., Elife. November 1, 2019; 8
A Xenopus oocyte model system to study action potentials. , Corbin-Leftwich A., J Gen Physiol. November 5, 2018; 150 (11): 1583-1593.
Phoneutria nigriventer Spider Toxin PnTx2-1 (δ-Ctenitoxin-Pn1a) Is a Modulator of Sodium Channel Gating. , Peigneur S., Toxins (Basel). August 21, 2018; 10 (9):
Improving the characterization of calcium channel gating pore currents with Stac3. , Chahine M., J Gen Physiol. March 5, 2018; 150 (3): 375-378.
Regulation of Na+ channel inactivation by the DIII and DIV voltage-sensing domains. , Hsu EJ ., J Gen Physiol. March 6, 2017; 149 (3): 389-403.
Ultrasound modulates ion channel currents. , Kubanek J., Sci Rep. April 26, 2016; 6 24170.
Splicing misregulation of SCN5A contributes to cardiac-conduction delay and heart arrhythmia in myotonic dystrophy. , Freyermuth F., Nat Commun. April 11, 2016; 7 11067.
Binary architecture of the Nav1.2-β2 signaling complex. , Das S., Elife. January 28, 2016; 5
Revealing the Function and the Structural Model of Ts4: Insights into the "Non-Toxic" Toxin from Tityus serrulatus Venom. , Pucca MB., Toxins (Basel). June 30, 2015; 7 (7): 2534-50.
The Scorpion Toxin Tf2 from Tityus fasciolatus Promotes Nav1.3 Opening. , Camargos TS., PLoS One. June 5, 2015; 10 (6): e0128578.
Xenopus borealis as an alternative source of oocytes for biophysical and pharmacological studies of neuronal ion channels. , Cristofori-Armstrong B., Sci Rep. January 12, 2015; 5 14763.
Nav1.1 modulation by a novel triazole compound attenuates epileptic seizures in rodents. , Gilchrist J., ACS Chem Biol. May 16, 2014; 9 (5): 1204-12.
Action of clathrodin and analogues on voltage-gated sodium channels. , Peigneur S., Mar Drugs. March 28, 2014; 12 (4): 2132-43.
Functional expression of Rat Nav1.6 voltage-gated sodium channels in HEK293 cells: modulation by the auxiliary β1 subunit. , He B., PLoS One. January 1, 2014; 9 (1): e85188.
Domain IV voltage-sensor movement is both sufficient and rate limiting for fast inactivation in sodium channels. , Capes DL., J Gen Physiol. August 1, 2013; 142 (2): 101-12.
Molecular mechanism of voltage sensing in voltage-gated proton channels. , Gonzalez C., J Gen Physiol. March 1, 2013; 141 (3): 275-85.
Multiple pore conformations driven by asynchronous movements of voltage sensors in a eukaryotic sodium channel. , Goldschen-Ohm MP., Nat Commun. January 1, 2013; 4 1350.
Intermediate state trapping of a voltage sensor. , Lacroix JJ., J Gen Physiol. December 1, 2012; 140 (6): 635-52.
Estimating the voltage-dependent free energy change of ion channels using the median voltage for activation. , Chowdhury S., J Gen Physiol. January 1, 2012; 139 (1): 3-17.
Functional properties and toxin pharmacology of a dorsal root ganglion sodium channel viewed through its voltage sensors. , Bosmans F., J Gen Physiol. July 1, 2011; 138 (1): 59-72.
Phyla- and Subtype-Selectivity of CgNa, a Na Channel Toxin from the Venom of the Giant Caribbean Sea Anemone Condylactis Gigantea. , Billen B., Front Pharmacol. November 23, 2010; 1 133.
Molecular mechanism of allosteric modification of voltage-dependent sodium channels by local anesthetics. , Arcisio-Miranda M., J Gen Physiol. November 1, 2010; 136 (5): 541-54.
Membrane trauma and Na+ leak from Nav1.6 channels. , Wang JA ., Am J Physiol Cell Physiol. October 1, 2009; 297 (4): C823-34.
The external pore loop interacts with S6 and S3-S4 linker in domain 4 to assume an essential role in gating control and anticonvulsant action in the Na(+) channel. , Yang YC ., J Gen Physiol. August 1, 2009; 134 (2): 95-113.
H+ pump-dependent changes in membrane voltage are an early mechanism necessary and sufficient to induce Xenopus tail regeneration. , Adams DS ., Development. April 1, 2007; 134 (7): 1323-35.
RE-1 silencer of transcription/neural restrictive silencer factor modulates ectodermal patterning during Xenopus development. , Olguín P., J Neurosci. March 8, 2006; 26 (10): 2820-9.
Occurrence of a tetrodotoxin-sensitive calcium current in rat ventricular myocytes after long-term myocardial infarction. , Alvarez JL., Cardiovasc Res. September 1, 2004; 63 (4): 653-61.
Repressor element-1 silencing transcription/ neuron-restrictive silencer factor is required for neural sodium channel expression during development of Xenopus. , Armisén R., J Neurosci. October 1, 2002; 22 (19): 8347-51.