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XB-ANAT-3713
Papers associated with left (and kmt2d)
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A convergent molecular network underlying autism and congenital heart disease., Rosenthal SB., Cell Syst. November 17, 2021; 12 (11): 1094-1107.e6. |
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The histone methyltransferase KMT2D, mutated in Kabuki syndrome patients, is required for neural crest cell formation and migration., Schwenty-Lara J., Hum Mol Genet. January 15, 2020; 29 (2): 305-319. |
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Loss of function of Kmt2d, a gene mutated in Kabuki syndrome, affects heart development in Xenopus laevis., Schwenty-Lara J., Dev Dyn. June 1, 2019; 248 (6): 465-476. |
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A phospho-dependent mechanism involving NCoR and KMT2D controls a permissive chromatin state at Notch target genes., Oswald F., Nucleic Acids Res. June 2, 2016; 44 (10): 4703-20. |
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WDR5 associates with histone H3 methylated at K4 and is essential for H3 K4 methylation and vertebrate development., Wysocka J., Cell. June 17, 2005; 121 (6): 859-72. |
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