Papers associated with left (and kcnd2)

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	S-glutathionylation of an auxiliary subunit confers redox sensitivity to Kv4 channel inactivation., Jerng HH., PLoS One. January 1, 2014; 9 (3): e93315.
	Impaired ion channel function related to a common KCNQ1 mutation - implications for risk stratification in long QT syndrome 1., Aidery P., Gene. December 10, 2012; 511 (1): 26-33.
	Modeling-independent elucidation of inactivation pathways in recombinant and native A-type Kv channels., Fineberg JD., J Gen Physiol. November 1, 2012; 140 (5): 513-27.
	Mutations in IRX5 impair craniofacial development and germ cell migration via SDF1., Bonnard C., Nat Genet. May 13, 2012; 44 (6): 709-13.
	Dynamic coupling of voltage sensor and gate involved in closed-state inactivation of kv4.2 channels., Barghaan J., J Gen Physiol. February 1, 2009; 133 (2): 205-24.
	Molecular and functional characterization of Kv4.2 and KChIP2 expressed in the porcine left ventricle., Schultz JH., Pflugers Arch. May 1, 2007; 454 (2): 195-207.
	Diminished Kv4.2/3 but not KChIP2 levels reduce the cardiac transient outward K+ current in spontaneously hypertensive rats., Goltz D., Cardiovasc Res. April 1, 2007; 74 (1): 85-95.
	Functionally active t1-t1 interfaces revealed by the accessibility of intracellular thiolate groups in kv4 channels., Wang G., J Gen Physiol. July 1, 2005; 126 (1): 55-69.

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