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Summary Anatomy Item Literature (4906) Expression Attributions Wiki
XB-ANAT-3713

Papers associated with left (and cacna1a)

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A convergent molecular network underlying autism and congenital heart disease., Rosenthal SB., Cell Syst. November 17, 2021; 12 (11): 1094-1107.e6.            

Functional assessment of the "two-hit" model for neurodevelopmental defects in Drosophila and X. laevis., Pizzo L., PLoS Genet. April 5, 2021; 17 (4): e1009112.                    

A Single Amino Acid Deletion (ΔF1502) in the S6 Segment of CaV2.1 Domain III Associated with Congenital Ataxia Increases Channel Activity and Promotes Ca2+ Influx., Bahamonde MI., PLoS One. January 1, 2015; 10 (12): e0146035.                

The inhibition of functional expression of calcium channels by prion protein demonstrates competition with α2δ for GPI-anchoring pathways., Alvarez-Laviada A., Biochem J. March 1, 2014; 458 (2): 365-74.          

N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2., Page KM., J Biol Chem. January 8, 2010; 285 (2): 835-44.              

Cloning and characterization of voltage-gated calcium channel alpha1 subunits in Xenopus laevis during development., Lewis BB., Dev Dyn. November 1, 2009; 238 (11): 2891-902.                                

Disruption of the IS6-AID linker affects voltage-gated calcium channel inactivation and facilitation., Findeisen F., J Gen Physiol. March 1, 2009; 133 (3): 327-43.              

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