Papers associated with left (and bax)

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	Time-resolved quantitative proteomic analysis of the developing Xenopus otic vesicle reveals putative congenital hearing loss candidates., Baxi AB., iScience. September 15, 2023; 26 (9): 107665.
	Thyroid hormone receptor knockout prevents the loss of Xenopus tail regeneration capacity at metamorphic climax., Wang S., Cell Biosci. February 23, 2023; 13 (1): 40.
	Thyroid Hormone Receptor Is Essential for Larval Epithelial Apoptosis and Adult Epithelial Stem Cell Development but Not Adult Intestinal Morphogenesis during Xenopus tropicalis Metamorphosis., Shibata Y., Cells. March 3, 2021; 10 (3):
	Evidence for Integrin - Venus Kinase Receptor 1 Alliance in the Ovary of Schistosoma mansoni Females Controlling Cell Survival., Gelmedin V., PLoS Pathog. January 23, 2017; 13 (1): e1006147.
	Early development of the neural plate: new roles for apoptosis and for one of its main effectors caspase-3., Juraver-Geslin HA., Genesis. February 1, 2015; 53 (2): 203-24.
	Visual and functional demonstration of growing Bax-induced pores in mitochondrial outer membranes., Gillies LA., Mol Biol Cell. January 15, 2015; 26 (2): 339-49.
	Polypyrimidine tract-binding protein is required for the repression of gene expression by all-trans retinoic acid., Tamanoue Y., Dev Growth Differ. June 1, 2010; 52 (5): 469-79.
	Hairy2 functions through both DNA-binding and non DNA-binding mechanisms at the neural plate border in Xenopus., Nichane M., Dev Biol. October 15, 2008; 322 (2): 368-80.
	Mitochondrial permeabilization relies on BH3 ligands engaging multiple prosurvival Bcl-2 relatives, not Bak., Uren RT., J Cell Biol. April 23, 2007; 177 (2): 277-87.
	Developmental cell death during Xenopus metamorphosis involves BID cleavage and caspase 2 and 8 activation., Du Pasquier D., Dev Dyn. August 1, 2006; 235 (8): 2083-94.
	Implication of bax in Xenopus laevis tail regression at metamorphosis., Sachs LM., Dev Dyn. December 1, 2004; 231 (4): 671-82.
	The midblastula transition in Xenopus embryos activates multiple pathways to prevent apoptosis in response to DNA damage., Finkielstein CV., Proc Natl Acad Sci U S A. January 30, 2001; 98 (3): 1006-11.
	The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol., Kluck RM., J Cell Biol. November 15, 1999; 147 (4): 809-22.

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